Neuropathic pain: role for presynaptic T-type channels in nociceptive signaling

Hydrogen sulfide-induced mechanical hyperalgesia and allodynia require activation of both Cav3.2 and TRPA1 channels in mice

(2012) Kazumasa Okubo et al.   BRITISH JOURNAL OF PHARMACOLOGY

Presynaptic CaV3.2 Channels Regulate Excitatory Neurotransmission in Nociceptive Dorsal Horn Neurons

(2012) M. O. Jacus et al.   JOURNAL OF NEUROSCIENCE

Diabetic neuropathy enhances voltage-activated Ca2+ channel activity and its control by M4 muscarinic receptors in primary sensory neurons

(2011) Xue-Hong Cao et al.   JOURNAL OF NEUROCHEMISTRY

Upregulation of Cav3.2 T-type calcium channels targeted by endogenous hydrogen sulfide contributes to maintenance of neuropathic pain

(2010) Tomoko Takahashi et al.   PAIN

Cellular and Molecular Mechanisms of Pain

(2009) Allan I. Basbaum et al.   CELL

Selective T-Type Calcium Channel Blockade Alleviates Hyperalgesia in ob/ob Mice

(2009) J. R. Latham et al.   DIABETES

In vivo silencing of the CaV3.2 T-type calcium channels in sensory neurons alleviates hyperalgesia in rats with streptozocin-induced diabetic neuropathy

(2009) Richard B. Messinger et al.   PAIN

Upregulation of the T-Type Calcium Current in Small Rat Sensory Neurons After Chronic Constrictive Injury of the Sciatic Nerve

(2008) Miljen M. Jagodic et al.   JOURNAL OF NEUROPHYSIOLOGY

Diabetic neuropathy: Mechanisms to management

(2008) James L. Edwards et al.   PHARMACOLOGY & THERAPEUTICS