Slc26a11 is prominently expressed in the brain and functions as a chloride channel: expression in Purkinje cells and stimulation of V H+-ATPase

Raising cytosolic Cl−in cerebellar granule cells affects their excitability and vestibulo-ocular learning

(2012) Patricia Seja et al.   EMBO JOURNAL

Novel Insights into the Pathomechanisms of Skeletal Muscle Channelopathies

(2011) James A. Burge et al.   Current Neurology and Neuroscience Reports

Two types of chloride transporters are required for GABAAreceptor-mediated inhibition inC. elegans

(2011) Andrew Bellemer et al.   EMBO JOURNAL

Chloride: The queen of electrolytes?

(2011) Kenrick Berend et al.   European Journal of Internal Medicine

Cl−concentration changes and desensitization of GABAAand glycine receptors

(2011) Urban Karlsson et al.   JOURNAL OF GENERAL PHYSIOLOGY

ClC-2 Channels Regulate Neuronal Excitability, Not Intracellular Chloride Levels

(2011) S. Ratte et al.   JOURNAL OF NEUROSCIENCE

Slc26a11, a chloride transporter, localizes with the vacuolar H + -ATPase of A-intercalated cells of the kidney

(2011) Jie Xu et al.   KIDNEY INTERNATIONAL

Spatiotemporal firing patterns in the cerebellum

(2011) Chris I. De Zeeuw et al.   NATURE REVIEWS NEUROSCIENCE

Novel Treatment Targets for Cerebral Edema

(2011) Brian P. Walcott et al.   Neurotherapeutics

Anoctamin 6 is an essential component of the outwardly rectifying chloride channel

(2011) J. R. Martins et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Vesicular ATPase Inserted into the Plasma Membrane of Motor Terminals by Exocytosis Alkalinizes Cytosolic pH and Facilitates Endocytosis

(2010) Zhongsheng Zhang et al.   NEURON

ClC transporters: discoveries and challenges in defining the mechanisms underlying function and regulation of ClC-5

(2010) Leigh Wellhauser et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

Cellular physiology of the renal H+ATPase

(2009) Katherine G Blake-Palmer et al.   CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION

Deletion of the Chloride Transporter Slc26a7 Causes Distal Renal Tubular Acidosis and Impairs Gastric Acid Secretion

(2009) Jie Xu et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

The little we know on the structure and machinery of V-ATPase

(2009) S. Saroussi et al.   JOURNAL OF EXPERIMENTAL BIOLOGY

Slc26a9—Anion Exchanger, Channel and Na+ Transporter

(2009) Min-Hwang Chang et al.   JOURNAL OF MEMBRANE BIOLOGY

Pathophysiology and puzzles of the volume-sensitive outwardly rectifying anion channel

(2009) Yasunobu Okada et al.   JOURNAL OF PHYSIOLOGY-LONDON

Synaptic inhibition of Purkinje cells mediates consolidation of vestibulo-cerebellar motor learning

(2009) Peer Wulff et al.   NATURE NEUROSCIENCE

The V-type H+-ATPase in vesicular trafficking: targeting, regulation and function

(2008) Vladimir Marshansky et al.   CURRENT OPINION IN CELL BIOLOGY

Diverse transport modes by the solute carrier 26 family of anion transporters

(2008) Ehud Ohana et al.   JOURNAL OF PHYSIOLOGY-LONDON

TMEM16A confers receptor-activated calcium-dependent chloride conductance

(2008) Young Duk Yang et al.   NATURE

Physiological roles of CLC Cl−/H+ exchangers in renal proximal tubules

(2008) Vanessa Plans et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

Vacuolar H+-ATPase—an enzyme for all seasons

(2008) Shai Saroussi et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

Deletion of the chloride transporter Slc26a9 causes loss of tubulovesicles in parietal cells and impairs acid secretion in the stomach

(2008) J. Xu et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA