Clinical applications of miRNAs in cardiac remodeling and heart failure
Published 2010 View Full Article
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Title
Clinical applications of miRNAs in cardiac remodeling and heart failure
Authors
Keywords
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Journal
Personalized Medicine
Volume 7, Issue 5, Pages 531-548
Publisher
Future Medicine Ltd
Online
2010-09-29
DOI
10.2217/pme.10.44
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Note: Only part of the references are listed.- MiRSNPs or MiR-polymorphisms, new players in microRNA mediated regulation of the cell: Introducing microRNA pharmacogenomics
- (2010) Prasun J. Mishra et al. CELL CYCLE
- MiR423-5p As a Circulating Biomarker for Heart Failure
- (2010) Anke J. Tijsen et al. CIRCULATION RESEARCH
- Circulating microRNA: a novel potential biomarker for early diagnosis of acute myocardial infarction in humans
- (2010) Guo-Kun Wang et al. EUROPEAN HEART JOURNAL
- miR-9 and NFATc3 Regulate Myocardin in Cardiac Hypertrophy
- (2010) Kun Wang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- MicroRNA expression in response to murine myocardial infarction: miR-21 regulates fibroblast metalloprotease-2 via phosphatase and tensin homologue
- (2009) Sashwati Roy et al. CARDIOVASCULAR RESEARCH
- Long-Term Cardiac-Targeted RNA Interference for the Treatment of Heart Failure Restores Cardiac Function and Reduces Pathological Hypertrophy
- (2009) Lennart Suckau et al. CIRCULATION
- Reciprocal Regulation of Myocardial microRNAs and Messenger RNA in Human Cardiomyopathy and Reversal of the microRNA Signature by Biomechanical Support
- (2009) Scot J. Matkovich et al. CIRCULATION
- MicroRNA-320 Is Involved in the Regulation of Cardiac Ischemia/Reperfusion Injury by Targeting Heat-Shock Protein 20
- (2009) Xiao-Ping Ren et al. CIRCULATION
- Reciprocal Regulation of MicroRNA-1 and Insulin-Like Growth Factor-1 Signal Transduction Cascade in Cardiac and Skeletal Muscle in Physiological and Pathological Conditions
- (2009) Leonardo Elia et al. CIRCULATION
- Downregulation of MiR-199a Derepresses Hypoxia-Inducible Factor-1α and Sirtuin 1 and Recapitulates Hypoxia Preconditioning in Cardiac Myocytes
- (2009) Shweta Rane et al. CIRCULATION RESEARCH
- miR-1 Overexpression Enhances Ca 2+ Release and Promotes Cardiac Arrhythmogenesis by Targeting PP2A Regulatory Subunit B56α and Causing CaMKII-Dependent Hyperphosphorylation of RyR2
- (2009) Dmitry Terentyev et al. CIRCULATION RESEARCH
- Loss of Cardiac microRNA-Mediated Regulation Leads to Dilated Cardiomyopathy and Heart Failure
- (2009) Prakash K. Rao et al. CIRCULATION RESEARCH
- MicroRNA-133a Protects Against Myocardial Fibrosis and Modulates Electrical Repolarization Without Affecting Hypertrophy in Pressure-Overloaded Adult Hearts
- (2009) Scot J. Matkovich et al. CIRCULATION RESEARCH
- Adaptive and Maladptive Effects of SMAD3 Signaling in the Adult Heart After Hemodynamic Pressure Overloading
- (2009) Vijay Divakaran et al. Circulation-Heart Failure
- Unique MicroRNA Profile in End-stage Heart Failure Indicates Alterations in Specific Cardiovascular Signaling Networks
- (2009) Sathyamangla V. Naga Prasad et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice
- (2009) Thomas E. Callis et al. JOURNAL OF CLINICAL INVESTIGATION
- MicroRNA-21 protects against the H2O2-induced injury on cardiac myocytes via its target gene PDCD4
- (2009) Yunhui Cheng et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- MicroRNA-1 Negatively Regulates Expression of the Hypertrophy-Associated Calmodulin and Mef2a Genes
- (2009) S. Ikeda et al. MOLECULAR AND CELLULAR BIOLOGY
- MicroRNA polymorphisms: the future of pharmacogenomics, molecular epidemiology and individualized medicine
- (2009) Prasun J Mishra et al. PHARMACOGENOMICS
- miR-23a functions downstream of NFATc3 to regulate cardiac hypertrophy
- (2009) Z. Lin et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Quantitative miRNA expression analysis: Comparing microarrays with next-generation sequencing
- (2009) H. Willenbrock et al. RNA
- Conditional Dicer Gene Deletion in the Postnatal Myocardium Provokes Spontaneous Cardiac Remodeling
- (2008) Paula A. da Costa Martins et al. CIRCULATION
- miR-133 and miR-30 Regulate Connective Tissue Growth Factor
- (2008) Rudy F. Duisters et al. CIRCULATION RESEARCH
- microRNA-133a regulates cardiomyocyte proliferation and suppresses smooth muscle gene expression in the heart
- (2008) N. Liu et al. GENES & DEVELOPMENT
- Application of massively parallel sequencing to microRNA profiling and discovery in human embryonic stem cells
- (2008) R. D. Morin et al. GENOME RESEARCH
- Down-regulation ofmiR-1/miR-133Contributes to Re-expression of Pacemaker Channel GenesHCN2andHCN4in Hypertrophic Heart
- (2008) Xiaobin Luo et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- miR-21: a small multi-faceted RNA
- (2008) Anna M. Krichevsky et al. JOURNAL OF CELLULAR AND MOLECULAR MEDICINE
- Changes in Regulatory MicroRNA Expression in Myocardium of Heart Failure Patients on Left Ventricular Assist Device Support
- (2008) Marguérite E.I. Schipper et al. JOURNAL OF HEART AND LUNG TRANSPLANTATION
- miRNA expression in the failing human heart: Functional correlates
- (2008) Carmen Sucharov et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts
- (2008) Thomas Thum et al. NATURE
- LNA-mediated microRNA silencing in non-human primates
- (2008) Joacim Elmén et al. NATURE
- Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis
- (2008) E. van Rooij et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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