Increased mitochondrial ROS formation by acetaminophen in human hepatic cells is associated with gene expression changes suggesting disruption of the mitochondrial electron transport chain

Title
Increased mitochondrial ROS formation by acetaminophen in human hepatic cells is associated with gene expression changes suggesting disruption of the mitochondrial electron transport chain
Authors
Keywords
Toxicogenomics, Acetaminophen, HepG2, Microarray, ESR, Mitochondrial ROS
Journal
TOXICOLOGY LETTERS
Volume 234, Issue 2, Pages 139-150
Publisher
Elsevier BV
Online
2015-02-20
DOI
10.1016/j.toxlet.2015.02.012

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