Loss of SHIP-1 protein expression in high-risk myelodysplastic syndromes is associated with miR-210 and miR-155

Essential Roles for the Tec Family Kinases Tec and Btk in M-CSF Receptor Signaling Pathways That Regulate Macrophage Survival

(2014) M. Melcher et al.   JOURNAL OF IMMUNOLOGY

The inositol phosphatase SHIP-1 is negatively regulated by Fli-1 and its loss accelerates leukemogenesis

(2010) G. K. Lakhanpal et al.   BLOOD

Tyrosine phosphorylation of SHIP promotes its proteasomal degradation

(2010) Jens Ruschmann et al.   EXPERIMENTAL HEMATOLOGY

Slowly Produced MicroRNAs Control Protein Levels

(2010) Zakary L. Whichard et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Hematopoiesis-related microRNA expression in myelodysplastic syndromes

(2010) Aina Pons et al.   LEUKEMIA & LYMPHOMA

Src homology 2 domain-containing inositol-5-phosphatase and CCAAT enhancer-binding protein   are targeted by miR-155 in B cells of E -MiR-155 transgenic mice

(2009) S. Costinean et al.   BLOOD

Aberrant overexpression of microRNAs activate AKT signaling via down-regulation of tumor suppressors in natural killer-cell lymphoma/leukemia

(2009) Y. Yamanaka et al.   BLOOD

The 2008 revision of the World Health Organization (WHO) classification of myeloid neoplasms and acute leukemia: rationale and important changes

(2009) J. W. Vardiman et al.   BLOOD

Onco-miR-155 targets SHIP1 to promote TNFα-dependent growth of B cell lymphomas

(2009) Irene M. Pedersen et al.   EMBO Molecular Medicine

Hypoxia-Inducible mir-210 Regulates Normoxic Gene Expression Involved in Tumor Initiation

(2009) Xin Huang et al.   MOLECULAR CELL

Silencing of microRNA-155 in mice during acute inflammatory response leads to derepression of c/ebp Beta and down-regulation of G-CSF

(2009) Jesper Worm et al.   NUCLEIC ACIDS RESEARCH

Inositol phosphatase SHIP1 is a primary target of miR-155

(2009) R. M. O'Connell et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

SHIP is required for a functional hematopoietic stem cell niche

(2008) A. L. Hazen et al.   BLOOD