The ATPase activity of Fml1 is essential for its roles in homologous recombination and DNA repair
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Title
The ATPase activity of Fml1 is essential for its roles in homologous recombination and DNA repair
Authors
Keywords
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Journal
NUCLEIC ACIDS RESEARCH
Volume 40, Issue 19, Pages 9584-9595
Publisher
Oxford University Press (OUP)
Online
2012-07-29
DOI
10.1093/nar/gks715
References
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Related references
Note: Only part of the references are listed.- Processing of DNA structures via DNA unwinding and branch migration by the S. cerevisiae Mph1 protein
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- (2009) George-Lucian Moldovan et al. Annual Review of Genetics
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- Fancm-deficient mice reveal unique features of Fanconi anemia complementation group M
- (2009) Sietske T. Bakker et al. HUMAN MOLECULAR GENETICS
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- FANCM of the Fanconi anemia core complex is required for both monoubiquitination and DNA repair
- (2008) Yutong Xue et al. HUMAN MOLECULAR GENETICS
- The FANCM Ortholog Fml1 Promotes Recombination at Stalled Replication Forks and Limits Crossing Over during DNA Double-Strand Break Repair
- (2008) Weili Sun et al. MOLECULAR CELL
- The Fanconi Anemia Protein FANCM Can Promote Branch Migration of Holliday Junctions and Replication Forks
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- FANCM and FAAP24 Function in ATR-Mediated Checkpoint Signaling Independently of the Fanconi Anemia Core Complex
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- Remodeling of DNA replication structures by the branch point translocase FANCM
- (2008) K. Gari et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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