Zinc-induced Dnmt1 expression involves antagonism between MTF-1 and nuclear receptor SHP
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Zinc-induced Dnmt1 expression involves antagonism between MTF-1 and nuclear receptor SHP
Authors
Keywords
-
Journal
NUCLEIC ACIDS RESEARCH
Volume 40, Issue 11, Pages 4850-4860
Publisher
Oxford University Press (OUP)
Online
2012-02-23
DOI
10.1093/nar/gks159
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Transcription factor Nrf2 regulates SHP and lipogenic gene expression in hepatic lipid metabolism
- (2010) Jiansheng Huang et al. AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY
- The metal-responsive transcription factor-1 protein is elevated in human tumors
- (2010) Yihui Shi et al. CANCER BIOLOGY & THERAPY
- Dysregulation of p53/Sp1 Control Leads to DNA Methyltransferase-1 Overexpression in Lung Cancer
- (2010) R.-K. Lin et al. CANCER RESEARCH
- BRCA1 affects global DNA methylation through regulation of DNMT1
- (2010) Vivek Shukla et al. CELL RESEARCH
- Down-regulated microRNA-152 induces aberrant DNA methylation in hepatitis B virus-related hepatocellular carcinoma by targeting DNA methyltransferase 1
- (2010) Jinfeng Huang et al. HEPATOLOGY
- hNaa10p contributes to tumorigenesis by facilitating DNMT1-mediated tumor suppressor gene silencing
- (2010) Chung-Fan Lee et al. JOURNAL OF CLINICAL INVESTIGATION
- MicroRNA-21 and MicroRNA-148a Contribute to DNA Hypomethylation in Lupus CD4+ T Cells by Directly and Indirectly Targeting DNA Methyltransferase 1
- (2010) W. Pan et al. JOURNAL OF IMMUNOLOGY
- Systems-level analysis of gene expression data revealed NR0B2/SHP as potential tumor suppressor in human liver cancer
- (2010) Yun-Yong Park et al. MOLECULES AND CELLS
- Zip4 (Slc39a4) Expression is Activated in Hepatocellular Carcinomas and Functions to Repress Apoptosis, Enhance Cell Cycle and Increase Migration
- (2010) Benjamin P. Weaver et al. PLoS One
- MicroRNA-206 Targetsnotch3, Activates Apoptosis, and Inhibits Tumor Cell Migration and Focus Formation
- (2009) Guisheng Song et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Nuclear Receptor SHP Activates miR-206 Expression via a Cascade Dual Inhibitory Mechanism
- (2009) Guisheng Song et al. PLoS One
- A Conserved Gene Structure and Expression Regulation of miR-433 and miR-127 in Mammals
- (2009) Guisheng Song et al. PLoS One
- Regulation of DNMT1 stability through SET7-mediated lysine methylation in mammalian cells
- (2009) P.-O. Esteve et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Epigenetic Inhibition of Nuclear Receptor Small Heterodimer Partner Is Associated With and Regulates Hepatocellular Carcinoma Growth
- (2008) Nan He et al. GASTROENTEROLOGY
- Loss of orphan receptor small heterodimer partner sensitizes mice to liver injury from obstructive cholestasis
- (2008) Young Joo Park et al. HEPATOLOGY
- Orphan receptor small heterodimer partner suppresses tumorigenesis by modulating cyclin D1 expression and cellular proliferation
- (2008) Yuxia Zhang et al. HEPATOLOGY
- Loss of small heterodimer partner expression in the liver protects against dyslipidemia
- (2008) Helen B. Hartman et al. JOURNAL OF LIPID RESEARCH
- Zinc-Induced Formation of a Coactivator Complex Containing the Zinc-Sensing Transcription Factor MTF-1, p300/CBP, and Sp1
- (2008) Y. Li et al. MOLECULAR AND CELLULAR BIOLOGY
- The lysine demethylase LSD1 (KDM1) is required for maintenance of global DNA methylation
- (2008) Jing Wang et al. NATURE GENETICS
- Transcriptional mechanism for the paired miR-433 and miR-127 genes by nuclear receptors SHP and ERRγ
- (2008) Guisheng Song et al. NUCLEIC ACIDS RESEARCH
Become a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get StartedAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started