Metnase promotes restart and repair of stalled and collapsed replication forks
Published 2010 View Full Article
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Title
Metnase promotes restart and repair of stalled and collapsed replication forks
Authors
Keywords
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Journal
NUCLEIC ACIDS RESEARCH
Volume 38, Issue 17, Pages 5681-5691
Publisher
Oxford University Press (OUP)
Online
2010-05-11
DOI
10.1093/nar/gkq339
References
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Related references
Note: Only part of the references are listed.- The transposase domain protein Metnase/SETMAR suppresses chromosomal translocations
- (2010) Justin Wray et al. CANCER GENETICS AND CYTOGENETICS
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- (2009) Helen E Bryant et al. EMBO JOURNAL
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- (2009) Justin Wray et al. PLoS One
- The MCM helicase: linking checkpoints to the replication fork: Figure 1
- (2008) Susan L. Forsburg BIOCHEMICAL SOCIETY TRANSACTIONS
- Distinct RAD51 Associations with RAD52 and BCCIP in Response to DNA Damage and Replication Stress
- (2008) J. Wray et al. CANCER RESEARCH
- Mechanisms of Dealing with DNA Damage-Induced Replication Problems
- (2008) Magda Budzowska et al. CELL BIOCHEMISTRY AND BIOPHYSICS
- Ino80 Chromatin Remodeling Complex Promotes Recovery of Stalled Replication Forks
- (2008) Kenji Shimada et al. CURRENT BIOLOGY
- The human set and transposase domain protein Metnase interacts with DNA Ligase IV and enhances the efficiency and accuracy of non-homologous end-joining
- (2008) Robert Hromas et al. DNA REPAIR
- Human Pso4 Is a Metnase (SETMAR)-binding Partner That Regulates Metnase Function in DNA Repair
- (2008) Brian D. Beck et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- ATR and H2AX Cooperate in Maintaining Genome Stability under Replication Stress
- (2008) Rebecca A. Chanoux et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- The SET and transposase domain protein Metnase enhances chromosome decatenation: regulation by automethylation
- (2008) E. A. Williamson et al. NUCLEIC ACIDS RESEARCH
- Regulation of proliferating cell nuclear antigen ubiquitination in mammalian cells
- (2008) A. Niimi et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Bloom's Syndrome Helicase and Mus81 are Required to Induce Transient Double-strand DNA Breaks in Response to DNA Replication Stress
- (2007) Tsutomu Shimura et al. JOURNAL OF MOLECULAR BIOLOGY
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