Journal
NEUROLOGY
Volume 75, Issue 7, Pages 654-658Publisher
LIPPINCOTT WILLIAMS & WILKINS
DOI: 10.1212/WNL.0b013e3181ed9eae
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Funding
- NIH [NINDS R01 NS054805, TW001140, TW008273, R21 AI072093, D43 TW006581, T35 AI065385, R01 HD059005, R01 AI087776]
- Gates Foundation
- Wellcome Trust
- OPTIMUS Foundation
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A single enhancing lesion in the brain parenchyma, also called an inflammatory granuloma, is a frequent neurologic diagnosis. One of the commonest causes of this lesion is human neurocysticercosis, the infection by the larvae of the pork tapeworm, Taenia solium. Following the demonstration that viable cysticercosis cysts survive in good conditions for several years in the human brain, single cysticercal granulomas have been consistently interpreted as representing late degeneration of a long-established parasite. On the basis of epidemiologic, clinical, and laboratory evidence detailed in this article, we hypothesize that in most cases these inflammatory lesions correspond to parasites that die in the early steps of infection, likely as the natural result of the host immunity overcoming mild infections. Neurology (R) 2010; 75: 654-658
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