Journal
NEUROBIOLOGY OF DISEASE
Volume 123, Issue -, Pages 100-109Publisher
ACADEMIC PRESS INC ELSEVIER SCIENCE
DOI: 10.1016/j.nbd.2018.08.006
Keywords
Neurodegeneration; Tauopathies; beta amyloid; Neuroinflammation; Traumatic brain injury; Post-traumatic epilepsy
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Funding
- NINDS NIH HHS [U54 NS100064] Funding Source: Medline
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In the quest for developing new therapeutic targets for post-traumatic epilepsies (PTE), identifying mechanisms relevant to development and progression of disease is critical. A growing body of literature suggests involvement of neurodegenerative mechanisms in the pathophysiology of acquired epilepsies, including following traumatic brain injury (TBI). In this review, we discuss the potential of some of these mechanisms to be targets for the development of a therapy against PTE.
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