Foamy macrophages and the progression of the human tuberculosis granuloma

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Immune and Inflammatory Mechanisms of Atherosclerosis

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Cholesterol Metabolism Increases the Metabolic Pool of Propionate inMycobacterium tuberculosis

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Mycobacterium tuberculosis evades macrophage defenses by inhibiting plasma membrane repair

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MARCO, TLR2, and CD14 Are Required for Macrophage Cytokine Responses to Mycobacterial Trehalose Dimycolate and Mycobacterium tuberculosis

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Mycobacterium tuberculosis blocks crosslinking of annexin-1 and apoptotic envelope formation on infected macrophages to maintain virulence

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Cytoplasmic lipid droplets are translocated into the lumen of the Chlamydia trachomatis parasitophorous vacuole

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Mycobacterial persistence requires the utilization of host cholesterol

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Foamy Macrophages from Tuberculous Patients' Granulomas Constitute a Nutrient-Rich Reservoir for M. tuberculosis Persistence

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Influence of low-density lipoprotein (LDL) receptor on lipid composition, inflammation and parasitism during Toxoplasma gondii infection

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