High-level inhibition of mitochondrial complexes III and IV is required to increase glutamate release from the nerve terminal

Decylubiquinone Increases Mitochondrial Function in Synaptosomes

(2010) Jayne E. Telford et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Age-related deficiencies in complex I endogenous substrate availability and reserve capacity of complex IV in cortical neuron electron transport

(2009) Torrie T. Jones et al.   BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS

Complex I Is Rate-limiting for Oxygen Consumption in the Nerve Terminal

(2009) Jayne E. Telford et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Age-related changes in H2O2 production and bioenergetics in rat brain synaptosomes

(2008) Seán M. Kilbride et al.   BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS

Respiratory-chain diseases related to complex III deficiency

(2008) Paule Bénit et al.   BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH

Brain mitochondrial dysfunction in aging

(2008) Alberto Boveris et al.   IUBMB LIFE

Partial inhibition of complex I activity increases Ca2+-independent glutamate release rates from depolarized synaptosomes

(2008) Sen M. Kilbride et al.   JOURNAL OF NEUROCHEMISTRY