Expression of human A53T alpha-synuclein in the rat substantia nigra using a novel AAV1/2 vector produces a rapidly evolving pathology with protein aggregation, dystrophic neurite architecture and nigrostriatal degeneration with potential to model the pathology of Parkinson's disease

Published 2010 View Full Article

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Journal

Molecular Neurodegeneration

Volume 5, Issue 1, Pages 43

Publisher

Springer Nature

Online

2010-10-29

DOI

10.1186/1750-1326-5-43

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