Epigenetic induction of tissue inhibitor of matrix metalloproteinase-3 by green tea polyphenols in breast cancer cells
Published 2014 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Epigenetic induction of tissue inhibitor of matrix metalloproteinase-3 by green tea polyphenols in breast cancer cells
Authors
Keywords
-
Journal
MOLECULAR CARCINOGENESIS
Volume 54, Issue 6, Pages 485-499
Publisher
Wiley
Online
2014-01-31
DOI
10.1002/mc.22121
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Cancer statistics, 2013
- (2013) Rebecca Siegel et al. CA-A CANCER JOURNAL FOR CLINICIANS
- Multifaceted role of EZH2 in breast and prostate tumorigenesis
- (2013) Gauri Deb et al. Epigenetics
- Heterogeneous epigenetic regulation ofTIMP3in prostate cancer
- (2012) Toshiaki Shinojima et al. Epigenetics
- Epigallocatechin gallate induce cell death and apoptosis in triple negative breast cancer cells Hs578T
- (2012) Cornelia Braicu et al. JOURNAL OF DRUG TARGETING
- The Role of EZH2 in the Regulation of the Activity of Matrix Metalloproteinases in Prostate Cancer Cells
- (2012) Yong Jae Shin et al. PLoS One
- Green tea polyphenols causes cell cycle arrest and apoptosis in prostate cancer cells by suppressing class I histone deacetylases
- (2011) V. S. Thakur et al. CARCINOGENESIS
- (-)-Epigallocatechin-3-gallate and DZNep reduce polycomb protein level via a proteasome-dependent mechanism in skin cancer cells
- (2011) S. R. Choudhury et al. CARCINOGENESIS
- The Chemopreventive and Chemotherapeutic Potentials of Tea Polyphenols
- (2011) Vijay S. Thakur et al. CURRENT PHARMACEUTICAL BIOTECHNOLOGY
- Epigallocatechin-3-gallate (EGCG) downregulates gelatinase-B (MMP-9) by involvement of FAK/ERK/NFκB and AP-1 in the human breast cancer cell line MDA-MB-231
- (2010) Triparna Sen et al. ANTI-CANCER DRUGS
- The tissue inhibitors of metalloproteinases (TIMPs): An ancient family with structural and functional diversity
- (2010) Keith Brew et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
- (–)-Epigallocatechin-3-gallate down-regulates EGFR, MMP-2, MMP-9 and EMMPRIN and inhibits the invasion of MCF-7 tamoxifen-resistant cells
- (2010) Fulvia Farabegoli et al. BIOSCIENCE REPORTS
- 5-azacytidine treatment reorganizes genomic histone modification patterns
- (2010) Vitalina M. Komashko et al. Epigenetics
- Inhibition of tumor growth and induction of apoptosis in prostate cancer cell lines by overexpression of tissue inhibitor of matrix metalloproteinase-3
- (2009) L Zhang et al. CANCER GENE THERAPY
- The Bmi-1 polycomb protein antagonizes the (-)-epigallocatechin-3-gallate-dependent suppression of skin cancer cell survival
- (2009) S. Balasubramanian et al. CARCINOGENESIS
- CDKN1C (p57KIP2) Is a Direct Target of EZH2 and Suppressed by Multiple Epigenetic Mechanisms in Breast Cancer Cells
- (2009) Xiaojing Yang et al. PLoS One
- Effects of green tea polyphenol on methylation status of RECK gene and cancer cell invasion in oral squamous cell carcinoma cells
- (2008) K Kato et al. BRITISH JOURNAL OF CANCER
- Tube Travel: The Role of Proteases in Individual and Collective Cancer Cell Invasion
- (2008) P. Friedl et al. CANCER RESEARCH
- The Role of Tissue Inhibitors of Metalloproteinases in Tumorigenesis and Metastasis
- (2008) William Cruz-Munoz et al. CRITICAL REVIEWS IN CLINICAL LABORATORY SCIENCES
- Enhancer of Zeste Homologue 2 (EZH2) Down-regulates RUNX3 by Increasing Histone H3 Methylation
- (2008) Satoshi Fujii et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Multifunctional effect of epigallocatechin-3-gallate (EGCG) in downregulation of gelatinase-A (MMP-2) in human breast cancer cell line MCF-7
- (2008) Triparna Sen et al. LIFE SCIENCES
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreFind the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
Search