Genistein inhibits hepatocellular carcinoma cell migration by reversing the epithelial-mesenchymal transition: Partial mediation by the transcription factor NFAT1
Published 2013 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Genistein inhibits hepatocellular carcinoma cell migration by reversing the epithelial-mesenchymal transition: Partial mediation by the transcription factor NFAT1
Authors
Keywords
-
Journal
MOLECULAR CARCINOGENESIS
Volume 54, Issue 4, Pages 301-311
Publisher
Wiley
Online
2013-11-15
DOI
10.1002/mc.22100
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Resveratrol Inhibits Proliferation and Induces Apoptosis through the Hedgehog Signaling Pathway in Pancreatic Cancer Cell
- (2012) Wenhui Mo et al. PANCREATOLOGY
- Salinomycin Inhibits Proliferation and Induces Apoptosis of Human Hepatocellular Carcinoma Cells In Vitro and In Vivo
- (2012) Fan Wang et al. PLoS One
- RETRACTED: Notch-1 induces epithelial–mesenchymal transition consistent with cancer stem cell phenotype in pancreatic cancer cells
- (2011) Bin Bao et al. CANCER LETTERS
- Calcineurin-NFAT Signaling Critically Regulates Early Lineage Specification in Mouse Embryonic Stem Cells and Embryos
- (2011) Xiang Li et al. Cell Stem Cell
- Cyclopamine Blocked the Growth of Colorectal Cancer SW116 Cells by Modulating Some Target Genes of Gli1 in vitro
- (2011) Jian-Ye Wu et al. HEPATO-GASTROENTEROLOGY
- Over-expression of FoxM1 leads to epithelial-mesenchymal transition and cancer stem cell phenotype in pancreatic cancer cells
- (2011) Bin Bao et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Interferon Regulatory Factor 2 Binding Protein 2 Is a New NFAT1 Partner and Represses Its Transcriptional Activity
- (2011) F. R. G. Carneiro et al. MOLECULAR AND CELLULAR BIOLOGY
- Inhibition of IGF-1 Signaling by Genistein: Modulation of E-Cadherin Expression and Downregulation of β-Catenin Signaling in Hormone Refractory PC-3 Prostate Cancer Cells
- (2011) Joomin Lee et al. NUTRITION AND CANCER-AN INTERNATIONAL JOURNAL
- NFATc1 Regulation of TRAIL Expression in Human Intestinal Cells
- (2011) Qingding Wang et al. PLoS One
- Inhibition of cancer cell invasion and metastasis by genistein
- (2010) Janet M. Pavese et al. CANCER AND METASTASIS REVIEWS
- Genistein potentiates the effect of arsenic trioxide against human hepatocellular carcinoma: Role of Akt and nuclear factor-κB
- (2010) Yong Ma et al. CANCER LETTERS
- Increased Expression of Cellular Repressor of E1A-Stimulated Gene (CREG) in Gastric Cancer Patients: A Mechanism of Proliferation and Metastasis in Cancer
- (2010) Ling Xu et al. DIGESTIVE DISEASES AND SCIENCES
- Genistein synergizes with arsenic trioxide to suppress human hepatocellular carcinoma
- (2009) Hongchi Jiang et al. CANCER SCIENCE
- Epithelial-Mesenchymal Transitions in Development and Disease
- (2009) Jean Paul Thiery et al. CELL
- Soy Food Consumption and Risk of Prostate Cancer: A Meta-Analysis of Observational Studies
- (2009) Ye Won Hwang et al. NUTRITION AND CANCER-AN INTERNATIONAL JOURNAL
- A novel anti-cancer effect of genistein: reversal of epithelial mesenchymal transition in prostate cancer cells1
- (2008) Lin-lin ZHANG et al. ACTA PHARMACOLOGICA SINICA
- Dietary Flavonoids and Colorectal Adenoma Recurrence in the Polyp Prevention Trial
- (2008) G. Bobe et al. CANCER EPIDEMIOLOGY BIOMARKERS & PREVENTION
- Soy isoflavone genistein upregulates epithelial adhesion molecule E-cadherin expression and attenuates -catenin signaling in mammary epithelial cells
- (2008) Y. Su et al. CARCINOGENESIS
- Diagnosis and Treatment of Hepatocellular Carcinoma
- (2008) Hashem B. El-Serag et al. GASTROENTEROLOGY
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started