BCR-ABL alternative splicing as a common mechanism for imatinib resistance: evidence from molecular dynamics simulations

Title
BCR-ABL alternative splicing as a common mechanism for imatinib resistance: evidence from molecular dynamics simulations
Authors
Keywords
-
Journal
MOLECULAR CANCER THERAPEUTICS
Volume 7, Issue 12, Pages 3834-3841
Publisher
American Association for Cancer Research (AACR)
Online
2008-12-04
DOI
10.1158/1535-7163.mct-08-0482

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