Ranolazine inhibits NaV1.5-mediated breast cancer cell invasiveness and lung colonization

Potent inhibition by ropivacaine of metastatic colon cancer SW620 cell invasion and Na V 1.5 channel function

(2014) D.T. Baptista-Hon et al.   BRITISH JOURNAL OF ANAESTHESIA

Ovarian cancer: Ion channel and aquaporin expression as novel targets of clinical potential

(2013) Julia Frede et al.   EUROPEAN JOURNAL OF CANCER

NaV1.5 Na+ channels allosterically regulate the NHE-1 exchanger and promote the activity of breast cancer cell invadopodia

(2013) L. Brisson et al.   JOURNAL OF CELL SCIENCE

Therapeutic potential for phenytoin: targeting Nav1.5 sodium channels to reduce migration and invasion in metastatic breast cancer

(2012) Ming Yang et al.   BREAST CANCER RESEARCH AND TREATMENT

Cancer Invasion and the Microenvironment: Plasticity and Reciprocity

(2011) Peter Friedl et al.   CELL

Overexpression of NaV1.6 channels is associated with the invasion capacity of human cervical cancer

(2011) Everardo Hernandez-Plata et al.   INTERNATIONAL JOURNAL OF CANCER

Voltage-Gated Na+ Channel SCN5A Is a Key Regulator of a Gene Transcriptional Network That Controls Colon Cancer Invasion

(2010) C. D. House et al.   CANCER RESEARCH

NaV1.5 enhances breast cancer cell invasiveness by increasing NHE1-dependent H+ efflux in caveolae

(2010) L Brisson et al.   ONCOGENE

Voltage-gated Sodium Channel Activity Promotes Cysteine Cathepsin-dependent Invasiveness and Colony Growth of Human Cancer Cells

(2009) Ludovic Gillet et al.   JOURNAL OF BIOLOGICAL CHEMISTRY