Reduced TOR signaling sustains hyphal development in Candida albicans by lowering Hog1 basal activity
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Title
Reduced TOR signaling sustains hyphal development in Candida albicans by lowering Hog1 basal activity
Authors
Keywords
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Journal
MOLECULAR BIOLOGY OF THE CELL
Volume 24, Issue 3, Pages 385-397
Publisher
American Society for Cell Biology (ASCB)
Online
2012-11-23
DOI
10.1091/mbc.e12-06-0477
References
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Related references
Note: Only part of the references are listed.- A GATA Transcription Factor Recruits Hda1 in Response to Reduced Tor1 Signaling to Establish a Hyphal Chromatin State in Candida albicans
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- (2011) Deborah A Hogan et al. CURRENT OPINION IN MICROBIOLOGY
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- (2011) Yu-Ting Chen et al. EUKARYOTIC CELL
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- (2011) Robbie Loewith et al. GENETICS
- The Candida albicans-Specific Gene EED1 Encodes a Key Regulator of Hyphal Extension
- (2011) Ronny Martin et al. PLoS One
- Hyphal Development in Candida albicans Requires Two Temporally Linked Changes in Promoter Chromatin for Initiation and Maintenance
- (2011) Yang Lu et al. PLOS BIOLOGY
- The Sko1 protein represses the yeast-to-hypha transition and regulates the oxidative stress response in Candida albicans
- (2010) Rebeca Alonso-Monge et al. FUNGAL GENETICS AND BIOLOGY
- Mds3 Regulates Morphogenesis in Candida albicans through the TOR Pathway
- (2010) L. F. Zacchi et al. MOLECULAR AND CELLULAR BIOLOGY
- Systematic screens of a Candida albicans homozygous deletion library decouple morphogenetic switching and pathogenicity
- (2010) Suzanne M Noble et al. NATURE GENETICS
- An Extensive Circuitry for Cell Wall Regulation in Candida albicans
- (2010) Jill R. Blankenship et al. PLoS Pathogens
- Hsp90 Orchestrates Temperature-Dependent Candida albicans Morphogenesis via Ras1-PKA Signaling
- (2009) Rebecca S. Shapiro et al. CURRENT BIOLOGY
- Identifying infection-associated genes ofCandida albicansin the postgenomic era
- (2009) Duncan Wilson et al. FEMS YEAST RESEARCH
- Candida albicansCyr1, Cap1 and G-actin form a sensor/effector apparatus for activating cAMP synthesis in hyphal growth
- (2009) Hao Zou et al. MOLECULAR MICROBIOLOGY
- A Phenotypic Profile of the Candida albicans Regulatory Network
- (2009) Oliver R. Homann et al. PLoS Genetics
- The Protein Kinase Tor1 Regulates Adhesin Gene Expression in Candida albicans
- (2009) Robert J. Bastidas et al. PLoS Pathogens
- Bacterial Peptidoglycan Triggers Candida albicans Hyphal Growth by Directly Activating the Adenylyl Cyclase Cyr1p
- (2008) Xiao-Li Xu et al. Cell Host & Microbe
- Nutritional control via Tor signaling in Saccharomyces cerevisiae
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- Nitrogen Catabolite Repression-Sensitive Transcription as a Readout of Tor Pathway Regulation: The Genetic Background, Reporter Gene and GATA Factor Assayed Determine the Outcomes
- (2008) I. Georis et al. GENETICS
- Role of the RAM Network in Cell Polarity and Hyphal Morphogenesis in Candida albicans
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- Regulation of the Candida albicans Cell Wall Damage Response by Transcription Factor Sko1 and PAS Kinase Psk1
- (2008) Jason M. Rauceo et al. MOLECULAR BIOLOGY OF THE CELL
- Efg1-mediated Recruitment of NuA4 to Promoters Is Required for Hypha-specific Swi/Snf Binding and Activation in Candida albicans
- (2008) Yang Lu et al. MOLECULAR BIOLOGY OF THE CELL
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