PERK Integrates Autophagy and Oxidative Stress Responses To Promote Survival during Extracellular Matrix Detachment
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Title
PERK Integrates Autophagy and Oxidative Stress Responses To Promote Survival during Extracellular Matrix Detachment
Authors
Keywords
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Journal
MOLECULAR AND CELLULAR BIOLOGY
Volume 31, Issue 17, Pages 3616-3629
Publisher
American Society for Microbiology
Online
2011-06-28
DOI
10.1128/mcb.05164-11
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Related references
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- PERK promotes cancer cell proliferation and tumor growth by limiting oxidative DNA damage
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- Malignant Precursor Cells Pre-Exist in Human Breast DCIS and Require Autophagy for Survival
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- Autophagy and tumorigenesis
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- The unfolded protein response protects human tumor cells during hypoxia through regulation of the autophagy genes MAP1LC3B and ATG5
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- Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment
- (2009) Zachary T. Schafer et al. NATURE
- Modeling Morphogenesis and Oncogenesis in Three-Dimensional Breast Epithelial Cultures
- (2008) Christy Hebner et al. Annual Review of Pathology-Mechanisms of Disease
- Extracellular matrix regulation of autophagy
- (2008) Rebecca Lock et al. CURRENT OPINION IN CELL BIOLOGY
- Basal Levels of eIF2α Phosphorylation Determine Cellular Antioxidant Status by Regulating ATF4 and xCT Expression
- (2008) Jan Lewerenz et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- PERK-dependent regulation of lipogenesis during mouse mammary gland development and adipocyte differentiation
- (2008) E. Bobrovnikova-Marjon et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Induction of Autophagy during Extracellular Matrix Detachment Promotes Cell Survival
- (2007) Christopher Fung et al. MOLECULAR BIOLOGY OF THE CELL
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