microRNA-200b and microRNA-200c promote colorectal cancer cell proliferation via targeting the reversion-inducing cysteine-rich protein with Kazal motifs
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
microRNA-200b and microRNA-200c promote colorectal cancer cell proliferation via targeting the reversion-inducing cysteine-rich protein with Kazal motifs
Authors
Keywords
-
Journal
RNA Biology
Volume 12, Issue 3, Pages 276-289
Publisher
Informa UK Limited
Online
2015-05-28
DOI
10.1080/15476286.2015.1017208
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- miR-21 Down-Regulation Suppresses Cell Growth, Invasion and Induces Cell Apoptosis by Targeting FASL, TIMP3, and RECK Genes in Esophageal Carcinoma
- (2013) Na Wang et al. DIGESTIVE DISEASES AND SCIENCES
- On Measuring miRNAs after Transient Transfection of Mimics or Antisense Inhibitors
- (2013) Daniel W. Thomson et al. PLoS One
- Predictions and estimations of colorectal cancer mortality, prevalence and incidence in Aragon, Spain, for the period 1998-2022
- (2013) Dyego Leandro Bezerra-de-Souza et al. REVISTA ESPANOLA DE ENFERMEDADES DIGESTIVAS
- miR-200c inhibits invasion and migration in human colon cancer cells SW480/620 by targeting ZEB1
- (2012) Mian Ling Chen et al. CLINICAL & EXPERIMENTAL METASTASIS
- MicroRNA-21 Expression is regulated by β-catenin/STAT3 Pathway and Promotes Glioma Cell Invasion by Direct Targeting RECK
- (2012) Lei Han et al. CNS Neuroscience & Therapeutics
- MicroRNA-200c modulates epithelial-to-mesenchymal transition (EMT) in human colorectal cancer metastasis
- (2012) Keun Hur et al. GUT
- Keratinization-associated miR-7 and miR-21 Regulate Tumor Suppressor Reversion-inducing Cysteine-rich Protein with Kazal Motifs (RECK) in Oral Cancer
- (2012) Hyun Min Jung et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- miR-21 may acts as an oncomir by targeting RECK, a matrix metalloproteinase regulator, in prostate cancer
- (2012) Sabrina Thalita Reis et al. BMC Urology
- Global cancer statistics
- (2011) Ahmedin Jemal et al. CA-A CANCER JOURNAL FOR CLINICIANS
- Involvement of the SKP2–p27KIP1 pathway in suppression of cancer cell proliferation by RECK
- (2011) Y Yoshida et al. ONCOGENE
- Skp2 knockout reduces cell proliferation and mouse body size: and prevents cancer?
- (2010) Liang Zhu CELL RESEARCH
- Hypoxia and RAS-signaling pathways converge on and cooperatively downregulate, the RECK tumor-suppressor protein through microRNAs
- (2010) F Loayza-Puch et al. ONCOGENE
- A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells
- (2008) Ulrike Burk et al. EMBO REPORTS
- The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2
- (2008) S.-M. Park et al. GENES & DEVELOPMENT
- RECK Forms Cowbell-shaped Dimers and Inhibits Matrix Metalloproteinase-catalyzed Cleavage of Fibronectin
- (2008) Akira Omura et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- The miR-200 Family Inhibits Epithelial-Mesenchymal Transition and Cancer Cell Migration by Direct Targeting of E-cadherin Transcriptional RepressorsZEB1andZEB2
- (2008) Manav Korpal et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- In Vitro Evidence Suggests That miR-133a-mediated Regulation of Uncoupling Protein 2 (UCP2) Is an Indispensable Step in Myogenic Differentiation
- (2008) Xi Chen et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1
- (2008) Philip A. Gregory et al. NATURE CELL BIOLOGY
- Micromanagers of malignancy: role of microRNAs in regulating metastasis
- (2008) Li Ma et al. TRENDS IN GENETICS
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started