miR-22 is down-regulated in gastric cancer, and its overexpression inhibits cell migration and invasion via targeting transcription factor Sp1
Published 2013 View Full Article
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Title
miR-22 is down-regulated in gastric cancer, and its overexpression inhibits cell migration and invasion via targeting transcription factor Sp1
Authors
Keywords
miR-22, Gastric cancer, Invasion, Migration, Sp1
Journal
MEDICAL ONCOLOGY
Volume 30, Issue 2, Pages -
Publisher
Springer Nature
Online
2013-03-25
DOI
10.1007/s12032-013-0542-7
References
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- Characterization of Basigin Isoforms and the Inhibitory Function of Basigin-3 in Human Hepatocellular Carcinoma Proliferation and Invasion
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- MicroRNA Control of Invasion and Metastasis Pathways
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- Differentiation-associated miR-22 represses Max expression and inhibits cell cycle progression
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- microRNA-22, downregulated in hepatocellular carcinoma and correlated with prognosis, suppresses cell proliferation and tumourigenicity
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- Transcription factor Sp1 regulates expression of cancer-associated molecule CD147 in human lung cancer
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- Promoter hypomethylation up-regulates CD147 expression through increasing Sp1 binding and associates with poor prognosis in human hepatocellular carcinoma
- (2010) Ling-Min Kong et al. JOURNAL OF CELLULAR AND MOLECULAR MEDICINE
- Tumor-suppressive microRNA-22 inhibits the transcription of E-box-containing c-Myc target genes by silencing c-Myc binding protein
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- Control of EVI-1 oncogene expression in metastatic breast cancer cells through microRNA miR-22
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- miR-22 Forms a Regulatory Loop in PTEN/AKT Pathway and Modulates Signaling Kinetics
- (2010) Nadav Bar et al. PLoS One
- CANCER: The Metastasis Cascade
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