Strain dependence of diet-induced NASH and liver fibrosis in obese mice is linked to diabetes and inflammatory phenotype
Published 2013 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Strain dependence of diet-induced NASH and liver fibrosis in obese mice is linked to diabetes and inflammatory phenotype
Authors
Keywords
-
Journal
LIVER INTERNATIONAL
Volume 34, Issue 7, Pages 1084-1093
Publisher
Wiley
Online
2013-10-12
DOI
10.1111/liv.12335
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Pharmacological cholesterol lowering reverses fibrotic NASH in obese, diabetic mice with metabolic syndrome
- (2013) Derrick M. Van Rooyen et al. JOURNAL OF HEPATOLOGY
- Dietary modification dampens liver inflammation and fibrosis in obesity-related fatty liver disease
- (2013) Claire Z. Larter et al. Obesity
- A truncating mutation of Alms1 reduces the number of hypothalamic neuronal cilia in obese mice
- (2012) Déborah Heydet et al. Developmental Neurobiology
- Role of Obesity and Lipotoxicity in the Development of Nonalcoholic Steatohepatitis: Pathophysiology and Clinical Implications
- (2012) Kenneth Cusi GASTROENTEROLOGY
- Association between diabetes, family history of diabetes, and risk of nonalcoholic steatohepatitis and fibrosis
- (2012) Rohit Loomba et al. HEPATOLOGY
- Curcumin protects against thioacetamide-induced hepatic fibrosis by attenuating the inflammatory response and inducing apoptosis of damaged hepatocytes
- (2012) Mu-En Wang et al. JOURNAL OF NUTRITIONAL BIOCHEMISTRY
- Physical Activity Recommendations, Exercise Intensity, and Histological Severity of Nonalcoholic Fatty Liver Disease
- (2011) Kristin D Kistler et al. AMERICAN JOURNAL OF GASTROENTEROLOGY
- Hepatic Free Cholesterol Accumulates in Obese, Diabetic Mice and Causes Nonalcoholic Steatohepatitis
- (2011) Derrick M. Van Rooyen et al. GASTROENTEROLOGY
- Coordinated improvement in glucose tolerance, liver steatosis and obesity-associated inflammation by cannabinoid 1 receptor antagonism in fat Aussie mice
- (2011) K S Bell-Anderson et al. INTERNATIONAL JOURNAL OF OBESITY
- Increases in p53 expression induce CTGF synthesis by mouse and human hepatocytes and result in liver fibrosis in mice
- (2011) Takahiro Kodama et al. JOURNAL OF CLINICAL INVESTIGATION
- Peroxisome proliferator-activated receptor-α agonist, Wy 14 643, improves metabolic indices, steatosis and ballooning in diabetic mice with non-alcoholic steatohepatitis
- (2011) Claire Z Larter et al. JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY
- Genome-Wide Association Study Identifies Variants Associated With Histologic Features of Nonalcoholic Fatty Liver Disease
- (2010) Naga Chalasani et al. GASTROENTEROLOGY
- Homozygosity for the patatin-like phospholipase-3/adiponutrin I148M polymorphism influences liver fibrosis in patients with nonalcoholic fatty liver disease
- (2010) Luca Valenti et al. HEPATOLOGY
- A fresh look at NASH pathogenesis. Part 1: The metabolic movers
- (2010) Claire Z Larter et al. JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY
- Diabetes is a progression factor for hepatic fibrosis in a high fat fed mouse obesity model of non-alcoholic steatohepatitis
- (2010) Lisa Lo et al. JOURNAL OF HEPATOLOGY
- Roles of adipose restriction and metabolic factors in progression of steatosis to steatohepatitis in obese, diabetic mice
- (2009) Claire Z Larter et al. JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY
- Systematic review of risk factors for fibrosis progression in non-alcoholic steatohepatitis
- (2009) Curtis K. Argo et al. JOURNAL OF HEPATOLOGY
- Novel Transcription Factor-Like Function of Human Matrix Metalloproteinase 3 Regulating the CTGF/CCN2 Gene
- (2008) T. Eguchi et al. MOLECULAR AND CELLULAR BIOLOGY
- Genetic variation in PNPLA3 confers susceptibility to nonalcoholic fatty liver disease
- (2008) Stefano Romeo et al. NATURE GENETICS
Find the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
SearchAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started