EVI-1 interacts with histone methyltransferases SUV39H1 and G9a for transcriptional repression and bone marrow immortalization

Pathogenetic significance of ecotropic viral integration site-1 in hematological malignancies

(2009) Susumu Goyama et al.   CANCER SCIENCE

High EVI1 levels predict adverse outcome in acute myeloid leukemia: prevalence of EVI1 overexpression and chromosome 3q26 abnormalities underestimated

(2008) S. Lugthart et al.   BLOOD

AML1 mutations induced MDS and MDS/AML in a mouse BMT model

(2008) N. Watanabe-Okochi et al.   BLOOD

Evi-1 promotes para-aortic splanchnopleural hematopoiesis through up-regulation of GATA-2 and repression of TGF-b signaling

(2008) Tomohiko Sato et al.   CANCER SCIENCE

Evi-1 Is a Critical Regulator for Hematopoietic Stem Cells and Transformed Leukemic Cells

(2008) Susumu Goyama et al.   Cell Stem Cell

A novel interaction between the proto-oncogene Evi1 and histone methyltransferases, SUV39H1 and G9a

(2008) Dominik Spensberger et al.   FEBS LETTERS

EVI1 recruits the histone methyltransferase SUV39H1 for transcription repression

(2008) Francesca Cattaneo et al.   JOURNAL OF CELLULAR BIOCHEMISTRY

AML1-Evi-1 specifically transforms hematopoietic stem cells through fusion of the entire Evi-1 sequence to AML1

(2008) M Takeshita et al.   LEUKEMIA

Significant increase of self-renewal in hematopoietic cells after forced expression of EVI1

(2007) Leopoldo Laricchia-Robbio et al.   BLOOD CELLS MOLECULES AND DISEASES