The Protective Effect of Epigallocatechin-3 Gallate on Ischemia/Reperfusion Injury in Isolated Rat Hearts: Anex vivoApproach

The past, the present and the future of experimental research on myocardial ischemia and protection

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Mitochondrial oxidant stress triggers cell death in simulated ischemia–reperfusion

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Polyphenol (-)-Epigallocatechin Gallate during Ischemia Limits Infarct Size Via Mitochondrial KATP Channel Activation in Isolated Rat Hearts

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Salidroside Attenuates Apoptosis in Ischemic Cardiomyocytes: a Mechanism Through a Mitochondria-Dependent Pathway

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Sulforaphane protects ischemic injury of hearts through antioxidant pathway and mitochondrial KATP channels

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Apoptotic and non-apoptotic programmed cardiomyocyte death in ventricular remodelling

(2008) G. W. Dorn   CARDIOVASCULAR RESEARCH

Positive inotropic effects of epigallocatechin-3-gallate (EGCG) involve activation of Na+/H+and Na+/Ca2+exchangers

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Mechanisms Underlying Acute Protection From Cardiac Ischemia-Reperfusion Injury

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