Ets-1 Is Required for the Activation of VEGFR3 during Latent Kaposi's Sarcoma-Associated Herpesvirus Infection of Endothelial Cells
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Title
Ets-1 Is Required for the Activation of VEGFR3 during Latent Kaposi's Sarcoma-Associated Herpesvirus Infection of Endothelial Cells
Authors
Keywords
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Journal
JOURNAL OF VIROLOGY
Volume 87, Issue 12, Pages 6758-6768
Publisher
American Society for Microbiology
Online
2013-04-04
DOI
10.1128/jvi.03241-12
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Note: Only part of the references are listed.- The KSHV viral IL-6 homolog is sufficient to induce blood to lymphatic endothelial cell differentiation
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- KSHV Induction of Angiogenic and Lymphangiogenic Phenotypes
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- Ets family members induce lymphangiogenesis through physical and functional interaction with Prox1
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- Deletion of Kaposi's Sarcoma-Associated Herpesvirus FLICE Inhibitory Protein, vFLIP, from the Viral Genome Compromises the Activation of STAT1-Responsive Cellular Genes and Spindle Cell Formation in Endothelial Cells
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- Transcription factor Ets-1 in cytokine and chemokine gene regulation
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- Ets1 and Ets2 are required for endothelial cell survival during embryonic angiogenesis
- (2009) G. Wei et al. BLOOD
- Ets-1 binds cooperatively to the palindromic Ets-binding sites in the p53 promoter
- (2008) David Baillat et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Kaposi's Sarcoma-Associated Herpesvirus Confers a Survival Advantage to Endothelial Cells
- (2008) L. Wang et al. CANCER RESEARCH
- Activation of theVEGFR1Chromatin Domain
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- The angiopoietin-2 gene of endothelial cells is up-regulated in hypoxia by a HIF binding site located in its first intron and by the central factors GATA-2 and Ets-1
- (2008) Marie-Pierre Simon et al. JOURNAL OF CELLULAR PHYSIOLOGY
- Activation of Akt through gp130 Receptor Signaling Is Required for Kaposi's Sarcoma-Associated Herpesvirus-Induced Lymphatic Reprogramming of Endothelial Cells
- (2008) V. A. Morris et al. JOURNAL OF VIROLOGY
- The Transcription Factor ETS-1: Its Role in Tumour Development and Strategies for its Inhibition
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