Protein phosphatase 2A impairs IFNα-induced antiviral activity against the hepatitis C virus through the inhibition of STAT1 tyrosine phosphorylation

Interferon-β and interferon-λ signaling is not affected by interferon-induced refractoriness to interferon-α in vivo

(2011) Zuzanna Makowska et al.   HEPATOLOGY

dsRNA sensors and plasmacytoid dendritic cells in host defense and autoimmunity

(2011) Yaming Wang et al.   IMMUNOLOGICAL REVIEWS

Sorafenib Inhibits STAT3 Activation to Enhance TRAIL-Mediated Apoptosis in Human Pancreatic Cancer Cells

(2010) S. Huang et al.   MOLECULAR CANCER THERAPEUTICS

Inactivation of Mammalian Target of Rapamycin Increases STAT1 Nuclear Content and Transcriptional Activity in α4- and Protein Phosphatase 2A-dependent Fashion

(2009) Jill A. Fielhaber et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

The Minimum Replication Origin of Merkel Cell Polyomavirus Has a Unique Large T-Antigen Loading Architecture and Requires Small T-Antigen Expression for Optimal Replication

(2009) H. J. Kwun et al.   JOURNAL OF VIROLOGY

PKR protein kinase is activated by hepatitis C virus and inhibits viral replication through translational control

(2009) Ju-Il Kang et al.   VIRUS RESEARCH

Cellular serine/threonine phosphatase activity during human cytomegalovirus infection

(2008) Morgan Hakki et al.   VIROLOGY