Expression of the Epigenetic factor BORIS (CTCFL) in the Human Genome
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Title
Expression of the Epigenetic factor BORIS (CTCFL) in the Human Genome
Authors
Keywords
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Journal
Journal of Translational Medicine
Volume 9, Issue 1, Pages 213
Publisher
Springer Nature
Online
2011-12-15
DOI
10.1186/1479-5876-9-213
References
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Related references
Note: Only part of the references are listed.- Susceptibility for breast cancer in young patients with short rare minisatellite alleles of BORIS
- (2011) Se-Lyun Yoon et al. BMB Reports
- Coordinated Cancer Germline Antigen Promoter and Global DNA Hypomethylation in Ovarian Cancer: Association with the BORIS/CTCF Expression Ratio and Advanced Stage
- (2011) A. Woloszynska-Read et al. CLINICAL CANCER RESEARCH
- CTCF: Master Weaver of the Genome
- (2009) Jennifer E. Phillips et al. CELL
- The Prioritization of Cancer Antigens: A National Cancer Institute Pilot Project for the Acceleration of Translational Research
- (2009) M. A. Cheever et al. CLINICAL CANCER RESEARCH
- Epigenetic Silencing of the p16INK4a Tumor Suppressor Is Associated with Loss of CTCF Binding and a Chromatin Boundary
- (2009) Michael Witcher et al. MOLECULAR CELL
- The pluripotency factor Oct4 interacts with Ctcf and also controls X-chromosome pairing and counting
- (2009) Mary E. Donohoe et al. NATURE
- Coordinated Activation of Candidate Proto-Oncogenes and Cancer Testes Antigens via Promoter Demethylation in Head and Neck Cancer and Lung Cancer
- (2009) Ian M. Smith et al. PLoS One
- Epigenetic regulation in mammalian preimplantation embryo development
- (2009) Lingjun Shi et al. Reproductive Biology and Endocrinology
- Selective apoptosis of breast cancer cells by siRNA targeting of BORIS
- (2008) Christopher J. Dougherty et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Differences in evolutionary pressure acting within highly conserved ortholog groups
- (2008) Teresa M Przytycka et al. BMC EVOLUTIONARY BIOLOGY
- BORIS, a paralogue of the transcription factor, CTCF, is aberrantly expressed in breast tumours
- (2008) V D'Arcy et al. BRITISH JOURNAL OF CANCER
- DNA Methyltransferase 1 and 3B Activate BAG-1 Expression via Recruitment of CTCFL/BORIS and Modulation of Promoter Histone Methylation
- (2008) L. Sun et al. CANCER RESEARCH
- CTCFL/BORIS Is a Methylation-Independent DNA-Binding Protein That Preferentially Binds to the Paternal H19 Differentially Methylated Region
- (2008) P. Nguyen et al. CANCER RESEARCH
- BAT3 and SET1A Form a Complex with CTCFL/BORIS To Modulate H3K4 Histone Dimethylation and Gene Expression
- (2008) P. Nguyen et al. MOLECULAR AND CELLULAR BIOLOGY
- Differential expression of the embryo/cancer gene ECSA(DPPA2), the cancer/testis gene BORIS and the pluripotency structural gene OCT4, in human preimplantation development
- (2008) M. Monk et al. MOLECULAR HUMAN REPRODUCTION
- Genome-wide identification of in vivo protein-DNA binding sites from ChIP-Seq data
- (2008) R. Jothi et al. NUCLEIC ACIDS RESEARCH
- The Evolution of Epigenetic Regulators CTCF and BORIS/CTCFL in Amniotes
- (2008) Timothy A. Hore et al. PLoS Genetics
- CTCF cis-Regulates Trinucleotide Repeat Instability in an Epigenetic Manner: A Novel Basis for Mutational Hot Spot Determination
- (2008) Randell T. Libby et al. PLoS Genetics
- Expression ofBORISin melanoma: Lack of association withMAGE-A1activation
- (2007) Olga Kholmanskikh et al. INTERNATIONAL JOURNAL OF CANCER
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