The Transmodulation of HER2 and EGFR by Substance P in Breast Cancer Cells Requires c-Src and Metalloproteinase Activation

Substance P Autocrine Signaling Contributes to Persistent HER2 Activation That Drives Malignant Progression and Drug Resistance in Breast Cancer

(2013) S. Garcia-Recio et al.   CANCER RESEARCH

Targeting of substance P induces cancer cell death and decreases the steady state of EGFR and Her2

(2011) Cristina Mayordomo et al.   JOURNAL OF CELLULAR PHYSIOLOGY

Tyrosine Kinase Receptor Transactivation Associated to G Protein-Coupled Receptors

(2010) Vanessa Almendro et al.   CURRENT DRUG TARGETS

Transactivation of ErbB-2 induced by tumor necrosis factor α promotes NF-κB activation and breast cancer cell proliferation

(2009) Martín A. Rivas et al.   BREAST CANCER RESEARCH AND TREATMENT

Expression of substance P and neurokinin-1-receptor in laryngeal cancer: linking chronic inflammation to cancer promotion and progression

(2008) Francisco Esteban et al.   HISTOPATHOLOGY

Persistent transactivation of EGFR and ErbB2/HER2 by protease-activated receptor-1 promotes breast carcinoma cell invasion

(2008) P Arora et al.   ONCOGENE

The NK-1 Receptor Is Expressed in Human Primary Gastric and Colon Adenocarcinomas and Is Involved in the Antitumor Action of L-733,060 and the Mitogenic Action of Substance P on Human Gastrointestinal Cancer Cell Lines

(2008) Marisa Rosso et al.   TUMOR BIOLOGY