Increased Expression of Angiogenic and Inflammatory Proteins in the Vitreous of Patients with Ischemic Central Retinal Vein Occlusion
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Increased Expression of Angiogenic and Inflammatory Proteins in the Vitreous of Patients with Ischemic Central Retinal Vein Occlusion
Authors
Keywords
Blood, Retinal vessels, Eyes, Inflammation, Surgical and invasive medical procedures, Atherosclerosis, Protein expression, Edema
Journal
PLoS One
Volume 10, Issue 5, Pages e0126859
Publisher
Public Library of Science (PLoS)
Online
2015-05-16
DOI
10.1371/journal.pone.0126859
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Ocular vascular occlusive disorders: Natural history of visual outcome
- (2014) Sohan Singh Hayreh PROGRESS IN RETINAL AND EYE RESEARCH
- Associations of matrix metalloproteinase-9 and monocyte chemoattractant protein-1 concentrations with carotid atherosclerosis, based on measurements of plaque and intima–media thickness
- (2013) Changhong Tan et al. ATHEROSCLEROSIS
- The 10-Year Incidence and Risk Factors of Retinal Vein Occlusion
- (2013) Jin Qiong Zhou et al. OPHTHALMOLOGY
- Efficacy and Safety of Intravitreal Therapy in Macular Edema Due to Branch and Central Retinal Vein Occlusion: a Systematic Review
- (2013) Amelie Pielen et al. PLoS One
- PENTRAXIN 3 AND OTHER INFLAMMATORY FACTORS IN CENTRAL RETINAL VEIN OCCLUSION AND MACULAR EDEMA
- (2013) Hidetaka Noma et al. RETINA-THE JOURNAL OF RETINAL AND VITREOUS DISEASES
- Osteoprotegerin as a Marker of Atherosclerosis in Diabetic Patients
- (2013) Areti Augoulea et al. International Journal of Endocrinology
- Intravitreal Aflibercept Injection for Macular Edema Secondary to Central Retinal Vein Occlusion: 1-Year Results From the Phase 3 COPERNICUS Study
- (2012) David M. Brown et al. AMERICAN JOURNAL OF OPHTHALMOLOGY
- ETS-1 Protein Regulates Vascular Endothelial Growth Factor-induced Matrix Metalloproteinase-9 and Matrix Metalloproteinase-13 Expression in Human Ovarian Carcinoma Cell Line SKOV-3
- (2012) Sonali Ghosh et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Benefit from Bevacizumab for Macular Edema in Central Retinal Vein Occlusion: Twelve-Month Results of a Prospective, Randomized Study
- (2012) David L. Epstein et al. OPHTHALMOLOGY
- A Longitudinal Analysis of Risk Factors Associated with Central Retinal Vein Occlusion
- (2012) Maxwell S. Stem et al. OPHTHALMOLOGY
- Comparison of cytokine levels from undiluted vitreous of untreated patients with retinal vein occlusion
- (2011) Michael J Koss et al. ACTA OPHTHALMOLOGICA
- Levels of VEGF but not VEGF 165b are Increased in the Vitreous of Patients With Retinal Vein Occlusion
- (2011) Christoph Ehlken et al. AMERICAN JOURNAL OF OPHTHALMOLOGY
- Matrix Metalloproteinase-10 Effectively Reduces Infarct Size in Experimental Stroke by Enhancing Fibrinolysis via a Thrombin-Activatable Fibrinolysis Inhibitor–Mediated Mechanism
- (2011) J. Orbe et al. CIRCULATION
- Expression profiles of cytokines and chemokines in vitreous fluid in diabetic retinopathy and central retinal vein occlusion
- (2011) Yukihiko Suzuki et al. JAPANESE JOURNAL OF OPHTHALMOLOGY
- Vitreous inflammatory factors and serous retinal detachment in central retinal vein occlusion: a case control series
- (2011) Hidetaka Noma et al. Journal of Inflammation-London
- Sustained Benefits from Ranibizumab for Macular Edema following Central Retinal Vein Occlusion: Twelve-Month Outcomes of a Phase III Study
- (2011) Peter A. Campochiaro et al. OPHTHALMOLOGY
- Regulation of the Ifng locus in the context of T-lineage specification and plasticity
- (2010) Anand Balasubramani et al. IMMUNOLOGICAL REVIEWS
- ATHEROSCLEROTIC AND THROMBOPHILIC RISK FACTORS IN PATIENTS WITH ISCHEMIC CENTRAL RETINAL VEIN OCCLUSION
- (2010) Andrea Sodi et al. RETINA-THE JOURNAL OF RETINAL AND VITREOUS DISEASES
- Matrix Metalloproteinase-10 Is Upregulated by Thrombin in Endothelial Cells and Increased in Patients With Enhanced Thrombin Generation
- (2009) Josune Orbe et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Differences in retinal neovascular tissue and vitreous humour in patients with type 1 and type 2 diabetes
- (2009) K Kinnunen et al. BRITISH JOURNAL OF OPHTHALMOLOGY
- Hypoxia Inducible Factor-1 Upregulates Adiponectin in Diabetic Mouse Hearts And Attenuates Post-Ischemic Injury
- (2009) Ramesh Natarajan et al. JOURNAL OF CARDIOVASCULAR PHARMACOLOGY
- Circulating Tissue Inhibitor of Matrix Metalloproteinase-4 (TIMP-4) in Systemic Sclerosis Patients with Elevated Pulmonary Arterial Pressure
- (2009) Gialafos J. Elias et al. MEDIATORS OF INFLAMMATION
- An integrative genomics approach identifies Hypoxia Inducible Factor-1 (HIF-1)-target genes that form the core response to hypoxia
- (2009) Yair Benita et al. NUCLEIC ACIDS RESEARCH
- The Prevalence of Retinal Vein Occlusion: Pooled Data from Population Studies from the United States, Europe, Asia, and Australia
- (2009) Sophie Rogers et al. OPHTHALMOLOGY
- Comprehensive Analysis of Inflammatory Immune Mediators in Vitreoretinal Diseases
- (2009) Takeru Yoshimura et al. PLoS One
- VEGF Stimulates HDAC7 Phosphorylation and Cytoplasmic Accumulation Modulating Matrix Metalloproteinase Expression and Angiogenesis
- (2008) Chang Hoon Ha et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Osteoprotegerin, vascular calcification and atherosclerosis
- (2008) Ann Van Campenhout et al. ATHEROSCLEROSIS
- The angiopoietin-2 gene of endothelial cells is up-regulated in hypoxia by a HIF binding site located in its first intron and by the central factors GATA-2 and Ets-1
- (2008) Marie-Pierre Simon et al. JOURNAL OF CELLULAR PHYSIOLOGY
- Role of metalloproteinases in platelet function
- (2007) María José Santos-Martínez et al. THROMBOSIS RESEARCH
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More