Inhibition of CXCL12/CXCR4 suppresses pulmonary arterial smooth muscle cell proliferation and cell cycle progression via PI3K/Akt pathway under hypoxia
Published 2014 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Inhibition of CXCL12/CXCR4 suppresses pulmonary arterial smooth muscle cell proliferation and cell cycle progression via PI3K/Akt pathway under hypoxia
Authors
Keywords
-
Journal
JOURNAL OF RECEPTORS AND SIGNAL TRANSDUCTION
Volume 35, Issue 4, Pages 329-339
Publisher
Informa UK Limited
Online
2014-11-25
DOI
10.3109/10799893.2014.984308
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Hypoxia downregulates PPARγ via an ERK1/2–NF-κB–Nox4-dependent mechanism in human pulmonary artery smooth muscle cells
- (2013) Xianghuai Lu et al. FREE RADICAL BIOLOGY AND MEDICINE
- Inhibition of Phosphatidylinositol 3-kinase/Akt Signaling Attenuates Hypoxia-induced Pulmonary Artery Remodeling and Suppresses CREB Depletion in Arterial Smooth Muscle Cells
- (2013) Chrystelle V. Garat et al. JOURNAL OF CARDIOVASCULAR PHARMACOLOGY
- Hypoxia induces connexin 43 dysregulation by modulating matrix metalloproteinases via MAPK signaling
- (2013) Xianghong Wu et al. MOLECULAR AND CELLULAR BIOCHEMISTRY
- Hypoxic Regulation of Pulmonary Vascular Smooth Muscle Cyclic Guanosine Monophosphate–Dependent Kinase by the Ubiquitin Conjugating System
- (2011) Ramaswamy Ramchandran et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- Key Role of 15-Lipoxygenase/15-Hydroxyeicosatetraenoic Acid in Pulmonary Vascular Remodeling and Vascular Angiogenesis Associated With Hypoxic Pulmonary Hypertension
- (2011) Cui Ma et al. HYPERTENSION
- Hypoxic Pulmonary Hypertension in Mice with Constitutively Active Platelet-Derived Growth Factor Receptor-β
- (2011) Bhola K. Dahal et al. Pulmonary Circulation
- Targeting of c-kit+ haematopoietic progenitor cells prevents hypoxic pulmonary hypertension
- (2010) N. Gambaryan et al. EUROPEAN RESPIRATORY JOURNAL
- SDF-1α/CXCR4 axis is involved in glucose-potentiated proliferation and chemotaxis in rat vascular smooth muscle cells
- (2010) Wei Jie et al. INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY
- Evidence of Dysfunction of Endothelial Progenitors in Pulmonary Arterial Hypertension
- (2009) Mark Toshner et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
- Dehydroepiandrosterone Reverses Systemic Vascular Remodeling Through the Inhibition of the Akt/GSK3-β/NFAT Axis
- (2009) Sébastien Bonnet et al. CIRCULATION
- Inhibition of the SDF-1/CXCR4 Axis Attenuates Neonatal Hypoxia-Induced Pulmonary Hypertension
- (2009) Karen C. Young et al. CIRCULATION RESEARCH
- 15-HETE suppresses K+ channel activity and inhibits apoptosis in pulmonary artery smooth muscle cells
- (2008) Yumei Li et al. APOPTOSIS
- Chemokines in Vascular Dysfunction and Remodeling
- (2008) Andreas Schober ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Statin ameliorates hypoxia-induced pulmonary hypertension associated with down-regulated stromal cell-derived factor-1
- (2008) K. Satoh et al. CARDIOVASCULAR RESEARCH
- Targeted Deletion of PTEN in Smooth Muscle Cells Results in Vascular Remodeling and Recruitment of Progenitor Cells Through Induction of Stromal Cell–Derived Factor-1α
- (2008) Raphael A. Nemenoff et al. CIRCULATION RESEARCH
- Subacute hypoxia suppresses Kv3.4 channel expression and whole-cell K+ currents through endogenous 15-hydroxyeicosatetraenoic acid in pulmonary arterial smooth muscle cells
- (2008) Lei Guo et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- A small molecule CXCR4 antagonist inhibits neointima formation and smooth muscle progenitor cell mobilization after arterial injury
- (2008) E. KARSHOVSKA et al. JOURNAL OF THROMBOSIS AND HAEMOSTASIS
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started