Stabilization of Kv4 protein by the accessory K+channel interacting protein 2 (KChIP2) subunit is required for the generation of native myocardial fast transient outward K+currents

Dissection of the voltage-activated potassium outward currents in adult mouse ventricular myocytes: I to,f, I to,s, I K,slow1, I K,slow2, and I ss

(2011) Jie Liu et al.   BASIC RESEARCH IN CARDIOLOGY

Repolarizing cardiac potassium channels: Multiple sites and mechanisms for CaMKII-mediated regulation

(2011) Jeanne M. Nerbonne   HEART RHYTHM

Co-assembly of Kv4 α Subunits with K+Channel-interacting Protein 2 Stabilizes Protein Expression and Promotes Surface Retention of Channel Complexes

(2010) Nicholas C. Foeger et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Interdependent Roles for Accessory KChIP2, KChIP3, and KChIP4 Subunits in the Generation of Kv4-Encoded IA Channels in Cortical Pyramidal Neurons

(2010) A. J. Norris et al.   JOURNAL OF NEUROSCIENCE

Neuronal voltage-gated K+ (Kv) channels function in macromolecular complexes

(2010) Aaron J. Norris et al.   NEUROSCIENCE LETTERS

Ancillary Subunits Associated With Voltage-Dependent K+ Channels

(2010) Olaf Pongs et al.   PHYSIOLOGICAL REVIEWS

Proteomic analysis highlights the molecular complexities of native Kv4 channel macromolecular complexes

(2010) Céline Marionneau et al.   SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY

Accessory Subunit KChIP2 Modulates the Cardiac L-Type Calcium Current

(2009) Morten B. Thomsen et al.   CIRCULATION RESEARCH

Molecular determinants of cardiac transient outward potassium current (Ito) expression and regulation

(2009) Noriko Niwa et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

Supramolecular Assemblies and Localized Regulation of Voltage-Gated Ion Channels

(2009) Shuiping Dai et al.   PHYSIOLOGICAL REVIEWS

Distinct Cellular and Molecular Mechanisms Underlie Functional Remodeling of Repolarizing K + Currents With Left Ventricular Hypertrophy

(2008) Céline Marionneau et al.   CIRCULATION RESEARCH

Targeted deletion of kcne2 impairs ventricular repolarization via disruption of IK,slow1 and Ito,f

(2008) Torsten K. Roepke et al.   FASEB JOURNAL

Deleting the accessory subunit KChIP2 results in loss of Ito,f and increased IK,slow that maintains normal action potential configuration

(2008) Morten B. Thomsen et al.   HEART RHYTHM

Translational control of localized mRNAs: restricting protein synthesis in space and time

(2008) Florence Besse et al.   NATURE REVIEWS MOLECULAR CELL BIOLOGY

Kv4.3 is not required for the generation of functional Ito,f channels in adult mouse ventricles

(2007) Noriko Niwa et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY