Modulation of contractile apparatus Ca2+sensitivity and disruption of excitation-contraction coupling byS-nitrosoglutathione in rat muscle fibres

Acute effects of reactive oxygen and nitrogen species on the contractile function of skeletal muscle

(2010) Graham D. Lamb et al.   JOURNAL OF PHYSIOLOGY-LONDON

Sequential effects of GSNO and H2O2 on the Ca2+ sensitivity of the contractile apparatus of fast- and slow-twitch skeletal muscle fibers from the rat

(2009) Timothy Spencer et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Myosin is reversibly inhibited by S-nitrosylation

(2009) Leonardo Nogueira et al.   BIOCHEMICAL JOURNAL

Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle

(2009) Andrew M Bellinger et al.   NATURE MEDICINE

Hydroxyl radical and glutathione interactions alter calcium sensitivity and maximum force of the contractile apparatus in rat skeletal muscle fibres

(2008) R. M. Murphy et al.   JOURNAL OF PHYSIOLOGY-LONDON

Calsequestrin content and SERCA determine normal and maximal Ca2+storage levels in sarcoplasmic reticulum of fast- and slow-twitch fibres of rat

(2008) Robyn M. Murphy et al.   JOURNAL OF PHYSIOLOGY-LONDON

Skeletal Muscle Fatigue: Cellular Mechanisms

(2008) D. G. Allen et al.   PHYSIOLOGICAL REVIEWS

Exercise-Induced Oxidative Stress: Cellular Mechanisms and Impact on Muscle Force Production

(2008) Scott K. Powers et al.   PHYSIOLOGICAL REVIEWS

Functional, structural, and chemical changes in myosin associated with hydrogen peroxide treatment of skeletal muscle fibers

(2007) Ewa Prochniewicz et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Reactive oxygen species and fatigue-induced prolonged low-frequency force depression in skeletal muscle fibres of rats, mice and SOD2 overexpressing mice

(2007) Joseph D. Bruton et al.   JOURNAL OF PHYSIOLOGY-LONDON

Chloride conductance in the transverse tubular system of rat skeletal muscle fibres: importance in excitation-contraction coupling and fatigue

(2007) T. L. Dutka et al.   JOURNAL OF PHYSIOLOGY-LONDON