Episodic spinal serotonin receptor activation elicits long-lasting phrenic motor facilitation by an NADPH oxidase-dependent mechanism

NADPH Oxidase Is Required for the Sensory Plasticity of the Carotid Body by Chronic Intermittent Hypoxia

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Reporting ethical matters inThe Journal of Physiology: standards and advice

(2009) Gordon B. Drummond   JOURNAL OF PHYSIOLOGY-LONDON

NADPH oxidase activity is necessary for acute intermittent hypoxia-induced phrenic long-term facilitation

(2009) P. M. MacFarlane et al.   JOURNAL OF PHYSIOLOGY-LONDON

Molecular biology of 5-HT receptors

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Hypoxia activates NADPH oxidase to increase [ROS]i and [Ca2+]i through the mitochondrial ROS-PKCɛ signaling axis in pulmonary artery smooth muscle cells

(2008) R RATHORE et al.   FREE RADICAL BIOLOGY AND MEDICINE

Developmental profiles of neurotransmitter receptors in respiratory motor nuclei

(2008) Leszek Kubin et al.   RESPIRATORY PHYSIOLOGY & NEUROBIOLOGY

Reactive oxygen species and respiratory plasticity following intermittent hypoxia

(2008) P.M. MacFarlane et al.   RESPIRATORY PHYSIOLOGY & NEUROBIOLOGY

Contributions of 5-HT neurons to respiratory control: Neuromodulatory and trophic effects

(2008) Matthew R. Hodges et al.   RESPIRATORY PHYSIOLOGY & NEUROBIOLOGY

Apocynin Is Not an Inhibitor of Vascular NADPH Oxidases but an Antioxidant

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Respiratory long-term facilitation following intermittent hypoxia requires reactive oxygen species formation

(2007) P.M. MacFarlane et al.   NEUROSCIENCE