Short-Chain Fatty Acids Activate AMP-Activated Protein Kinase and Ameliorate Ethanol-Induced Intestinal Barrier Dysfunction in Caco-2 Cell Monolayers
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Title
Short-Chain Fatty Acids Activate AMP-Activated Protein Kinase and Ameliorate Ethanol-Induced Intestinal Barrier Dysfunction in Caco-2 Cell Monolayers
Authors
Keywords
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Journal
JOURNAL OF NUTRITION
Volume 143, Issue 12, Pages 1872-1881
Publisher
Oxford University Press (OUP)
Online
2013-10-17
DOI
10.3945/jn.113.179549
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Note: Only part of the references are listed.- Butyrate Enhances Intestinal Epithelial Barrier Function via Up-Regulation of Tight Junction Protein Claudin-1 Transcription
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- Lactobacillus GG treatment ameliorates alcohol-induced intestinal oxidative stress, gut leakiness, and liver injury in a rat model of alcoholic steatohepatitis
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- Alcohol Stimulates Activation of Snail, Epidermal Growth Factor Receptor Signaling, and Biomarkers of EpithelialâMesenchymal Transition in Colon and Breast Cancer Cells
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- Metabolic Stress Evokes Decreases in Epithelial Barrier Function
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- Butyrate Enhances the Intestinal Barrier by Facilitating Tight Junction Assembly via Activation of AMP-Activated Protein Kinase in Caco-2 Cell Monolayers
- (2009) Luying Peng et al. JOURNAL OF NUTRITION
- Oats Supplementation Prevents Alcohol-Induced Gut Leakiness in Rats by Preventing Alcohol-Induced Oxidative Tissue Damage
- (2009) Y. Tang et al. JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS
- Butyrate protects Caco-2 cells from Campylobacter jejuni invasion and translocation
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- Evidence that chronic alcohol exposure promotes intestinal oxidative stress, intestinal hyperpermeability and endotoxemia prior to development of alcoholic steatohepatitis in rats
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