Physiological interactions between Nav1.7 and Nav1.8 sodium channels: a computer simulation study

Sodium-Calcium Exchanger and Multiple Sodium Channel Isoforms in Intra-Epidermal Nerve Terminals

(2016) Anna-Karin Persson et al.   Molecular Pain

ERK1/2 Mitogen-Activated Protein Kinase Phosphorylates Sodium Channel Nav1.7 and Alters Its Gating Properties

(2010) S. Stamboulian et al.   JOURNAL OF NEUROSCIENCE

Increased Peripheral Nerve Excitability and Local Nav1.8 RNA Up-Regulation in Painful Neuropathy

(2009) Devang Kashyap Thakor et al.   Molecular Pain

Multiple sodium channel isoforms and mitogen-activated protein kinases are present in painful human neuromas

(2008) Joel A. Black et al.   ANNALS OF NEUROLOGY

Spontaneous action potential generation due to persistent sodium channel currents in simulated carotid body afferent fibers

(2008) David F. Donnelly   JOURNAL OF APPLIED PHYSIOLOGY

Phosphorylation of Sodium Channel Nav1.8 by p38 Mitogen-Activated Protein Kinase Increases Current Density in Dorsal Root Ganglion Neurons

(2008) A. Hudmon et al.   JOURNAL OF NEUROSCIENCE

Nav1.7 Expression is Increased in Painful Human Dental Pulp

(2008) Songjiang Luo et al.   Molecular Pain

Changes in the expression of NaV1.7, NaV1.8 and NaV1.9 in a distinct population of dorsal root ganglia innervating the rat knee joint in a model of chronic inflammatory joint pain

(2007) Iain T. Strickland et al.   EUROPEAN JOURNAL OF PAIN

Subthreshold oscillations facilitate neuropathic spike discharge by overcoming membrane accommodation

(2007) Yifat Kovalsky et al.   EXPERIMENTAL NEUROLOGY