Differential Relationships of Reactive Astrocytes and Microglia to Fibrillar Amyloid Deposits in Alzheimer Disease
Published 2013 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Differential Relationships of Reactive Astrocytes and Microglia to Fibrillar Amyloid Deposits in Alzheimer Disease
Authors
Keywords
-
Journal
JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY
Volume 72, Issue 6, Pages 462-471
Publisher
Oxford University Press (OUP)
Online
2013-05-08
DOI
10.1097/nen.0b013e3182933788
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Faddish Stuff: Epigenetics and the Inheritance of Acquired Characteristics
- (2013) Mark Ptashne FASEB JOURNAL
- Neuropathological Alterations in Alzheimer Disease
- (2013) A. Serrano-Pozo et al. Cold Spring Harbor Perspectives in Medicine
- Apolipoprotein E4 effects in Alzheimer’s disease are mediated by synaptotoxic oligomeric amyloid-β
- (2012) Robert M. Koffie et al. BRAIN
- APOE genotype alters glial activation and loss of synaptic markers in mice
- (2012) Yuangui Zhu et al. GLIA
- Stable Size Distribution of Amyloid Plaques Over the Course of Alzheimer Disease
- (2012) Alberto Serrano-Pozo et al. JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY
- Reactive Glia not only Associates with Plaques but also Parallels Tangles in Alzheimer's Disease
- (2011) Alberto Serrano-Pozo et al. AMERICAN JOURNAL OF PATHOLOGY
- Apolipoprotein E isoform-dependent microglia migration
- (2011) Eiron Cudaback et al. FASEB JOURNAL
- Dynamic Analysis of Amyloid -Protein in Behaving Mice Reveals Opposing Changes in ISF versus Parenchymal A during Age-Related Plaque Formation
- (2011) S. Hong et al. JOURNAL OF NEUROSCIENCE
- Critical Role of Astroglial Apolipoprotein E and Liver X Receptor- Expression for Microglial A Phagocytosis
- (2011) D. Terwel et al. JOURNAL OF NEUROSCIENCE
- Neuronal and Axonal Loss Are Selectively Linked to Fibrillar Amyloid-β within Plaques of the Aged Primate Cerebral Cortex
- (2010) Palak Shah et al. AMERICAN JOURNAL OF PATHOLOGY
- Beneficial effect of human anti-amyloid-β active immunization on neurite morphology and tau pathology
- (2010) Alberto Serrano-Pozo et al. BRAIN
- CX3CR1 in Microglia Regulates Brain Amyloid Deposition through Selective Protofibrillar Amyloid- Phagocytosis
- (2010) Z. Liu et al. JOURNAL OF NEUROSCIENCE
- Amyloid Induces the Morphological Neurodegenerative Triad of Spine Loss, Dendritic Simplification, and Neuritic Dystrophies through Calcineurin Activation
- (2010) H.-Y. Wu et al. JOURNAL OF NEUROSCIENCE
- Locus ceruleus controls Alzheimer's disease pathology by modulating microglial functions through norepinephrine
- (2010) M. T. Heneka et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Macrophage-Mediated Degradation of -Amyloid via an Apolipoprotein E Isoform-Dependent Mechanism
- (2009) L. Zhao et al. JOURNAL OF NEUROSCIENCE
- Microglial VEGF Receptor Response Is an Integral Chemotactic Component in Alzheimer's Disease Pathology
- (2009) J. K. Ryu et al. JOURNAL OF NEUROSCIENCE
- Oligomeric amyloid associates with postsynaptic densities and correlates with excitatory synapse loss near senile plaques
- (2009) R. M. Koffie et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Dynamics of the Microglial/Amyloid Interaction Indicate a Role in Plaque Maintenance
- (2008) T. Bolmont et al. JOURNAL OF NEUROSCIENCE
- Microglial Dysfunction and Defective -Amyloid Clearance Pathways in Aging Alzheimer's Disease Mice
- (2008) S. E. Hickman et al. JOURNAL OF NEUROSCIENCE
- Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory
- (2008) Ganesh M Shankar et al. NATURE MEDICINE
- ApoE Promotes the Proteolytic Degradation of Aβ
- (2008) Qingguang Jiang et al. NEURON
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More