CaMKII inhibition with KN93 attenuates endothelin and serotonin receptor-mediated vasoconstriction and prevents subarachnoid hemorrhage-induced deficits in sensorimotor function
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Title
CaMKII inhibition with KN93 attenuates endothelin and serotonin receptor-mediated vasoconstriction and prevents subarachnoid hemorrhage-induced deficits in sensorimotor function
Authors
Keywords
CaMKII, Delayed cerebral ischemia, DREAM, Endothelin receptors, KN93, Serotonin receptors, Subarachnoid hemorrhage, Vasospasm
Journal
Journal of Neuroinflammation
Volume 11, Issue 1, Pages -
Publisher
Springer Nature
Online
2014-12-10
DOI
10.1186/s12974-014-0207-2
References
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- Randomized Trial of Clazosentan in Patients With Aneurysmal Subarachnoid Hemorrhage Undergoing Endovascular Coiling
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- Improvement in neurological outcome and abolition of cerebrovascular endothelin B and 5-hydroxytryptamine 1B receptor upregulation through mitogen-activated protein kinase kinase 1/2 inhibition after subarachnoid hemorrhage in rats
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- Enhanced expressions of microvascular smooth muscle receptors after focal cerebral ischemia occur via the MAPK MEK/ERK pathway
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- Pathogenesis of cerebral vasospasm following aneurysmal subarachnoid hemorrhage: Putative mechanisms and novel approaches
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- The non-excitable smooth muscle: Calcium signaling and phenotypic switching during vascular disease
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