N-Acetylaspartylglutamate is an agonist at mGluR3 in vivo and in vitro

Endogenous N-Acetylaspartylglutamate (NAAG) Inhibits Synaptic Plasticity/Transmission in the Amygdala in a Mouse Inflammatory Pain Model

(2016) Mary O Adedoyin et al.   Molecular Pain

N-Acetylaspartylglutamate Synthetase II SynthesizesN-Acetylaspartylglutamylglutamate

(2011) Julia Lodder-Gadaczek et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Advances in understanding the peptide neurotransmitter NAAG and appearance of a new member of the NAAG neuropeptide family

(2011) Joseph H. Neale et al.   JOURNAL OF NEUROCHEMISTRY

Metabotropic Glutamate Receptors: Physiology, Pharmacology, and Disease

(2010) Colleen M. Niswender et al.   Annual Review of Pharmacology and Toxicology

Sensing G protein-coupled receptor activation

(2010) Manuela Ambrosio et al.   NEUROPHARMACOLOGY

Glutamatergic Inhibition in Sensory Neocortex

(2009) C. C. Lee et al.   CEREBRAL CORTEX

The Neuroactive Peptide N-Acetylaspartylglutamate Is Not an Agonist at the Metabotropic Glutamate Receptor Subtype 3 of Metabotropic Glutamate Receptor

(2009) M. Chopra et al.   JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS

Effects of N-acetylaspartylglutamate (NAAG) at group II mGluRs and NMDAR

(2009) Anne-Cécile Fricker et al.   NEUROPHARMACOLOGY

Intracerebroventricular Administration of N-Acetylaspartylglutamate (NAAG) Peptidase Inhibitors is Analgesic in Inflammatory Pain

(2008) Tatsuo Yamamoto et al.   Molecular Pain