Pseudoprogression in patients with malignant gliomas treated with concurrent temozolomide and radiotherapy: potential role of p53

Association BetweenMGMTPromoter Hypermethylation and p53 Mutation in Glioblastoma

(2009) Jamal Shamsara et al.   CANCER INVESTIGATION

Brain tumor hypoxia: tumorigenesis, angiogenesis, imaging, pseudoprogression, and as a therapeutic target

(2009) Randy L. Jensen   JOURNAL OF NEURO-ONCOLOGY

p53 Mutant Human Glioma Cells Are Sensitive to UV-C-Induced Apoptosis Due to Impaired Cyclobutane Pyrimidine Dimer Removal

(2009) L. F.Z. Batista et al.   MOLECULAR CANCER RESEARCH

Brain Tumor Imaging in Clinical Trials

(2008) J.W. Henson et al.   AMERICAN JOURNAL OF NEURORADIOLOGY

Incidence of early pseudo‐progression in a cohort of malignant glioma patients treated with chemoirradiation with temozolomide

(2008) Walter Taal et al.   CANCER

High incidence of MGMT promoter methylation in primary glioblastomas without correlation with TP53 gene mutations

(2008) Emilia Jesien-Lewandowicz et al.   CANCER GENETICS AND CYTOGENETICS

MGMTPromoter Methylation Status Can Predict the Incidence and Outcome of Pseudoprogression After Concomitant Radiochemotherapy in Newly Diagnosed Glioblastoma Patients

(2008) Alba A. Brandes et al.   JOURNAL OF CLINICAL ONCOLOGY

Clinical features, mechanisms, and management of pseudoprogression in malignant gliomas

(2008) Dieta Brandsma et al.   LANCET ONCOLOGY