Increased sarcolipin expression and decreased sarco(endo)plasmic reticulum Ca2+ uptake in skeletal muscles of mouse models of Duchenne muscular dystrophy

Sarcoplasmic reticulum Ca2+permeation explored from the lumen side inmdxmuscle fibers under voltage control

(2012) Gaëlle Robin et al.   JOURNAL OF GENERAL PHYSIOLOGY

Hsp72 preserves muscle function and slows progression of severe muscular dystrophy

(2012) Stefan M. Gehrig et al.   NATURE

Enhanced Ca2+ transport and muscle relaxation in skeletal muscle from sarcolipin-null mice

(2011) A. Russell Tupling et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Mitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle

(2011) Sanjeewa A. Goonasekera et al.   JOURNAL OF CLINICAL INVESTIGATION

Excitation-contraction coupling alterations in mdx and utrophin/dystrophin double knockout mice: a comparative study

(2010) Joana Capote et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Upregulation of store-operated Ca2+ entry in dystrophic mdx mouse muscle

(2010) Joshua N. Edwards et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Review of Duchenne Muscular Dystrophy (DMD) for the Pediatricians in the Community

(2010) Sumit Verma et al.   CLINICAL PEDIATRICS

Leaky RyR2 trigger ventricular arrhythmias in Duchenne muscular dystrophy

(2010) J. Fauconnier et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Making Fast-Twitch Dystrophic Muscles Bigger Protects Them from Contraction Injury and Attenuates the Dystrophic Pathology

(2009) Stefan M. Gehrig et al.   AMERICAN JOURNAL OF PATHOLOGY

The decay phase of Ca2+ transients in skeletal muscle: regulation and physiologyThis paper is one of a selection of papers published in this Special Issue, entitled 14th International Biochemistry of Exercise Conference – Muscles as Molecular and Metabolic Machines, and has undergone the Journal’s usual peer review process.

(2009) A. Russell Tupling   Applied Physiology Nutrition and Metabolism

Sarcoplasmic-endoplasmic-reticulum Ca2+-ATPase and calsequestrin are overexpressed in spared intrinsic laryngeal muscles of dystrophin-deficientmdxmice

(2009) Renato Ferretti et al.   MUSCLE & NERVE

Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle

(2009) Andrew M Bellinger et al.   NATURE MEDICINE

Muscle genome-wide expression profiling during disease evolution in mdx mice

(2009) Mario Marotta et al.   PHYSIOLOGICAL GENOMICS