MiR-133b Contributes to Arsenic-Induced Apoptosis in U251 Glioma Cells by Targeting the hERG Channel
Published 2014 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
MiR-133b Contributes to Arsenic-Induced Apoptosis in U251 Glioma Cells by Targeting the hERG Channel
Authors
Keywords
miR-133b, miR-34a, hERG, Arsenic trioxide, Glioma
Journal
JOURNAL OF MOLECULAR NEUROSCIENCE
Volume 55, Issue 4, Pages 985-994
Publisher
Springer Nature
Online
2014-10-29
DOI
10.1007/s12031-014-0455-8
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Role of microRNAs in mechanisms of glioblastoma resistance to radio- and chemotherapy
- (2013) Ph. A. Koshkin et al. BIOCHEMISTRY-MOSCOW
- Understanding high grade glioma: Molecular mechanism, therapy and comprehensive management
- (2013) Yongzhi Wang et al. CANCER LETTERS
- Cathepsin L silencing enhances arsenic trioxide mediated in vitro cytotoxicity and apoptosis in glioblastoma U87MG spheroids
- (2013) Monika Primon et al. EXPERIMENTAL CELL RESEARCH
- Novel approaches to glioma drug design and drug screening
- (2013) Shivaputra A Patil et al. Expert Opinion on Drug Discovery
- A phase I trial of arsenic trioxide chemoradiotherapy for infiltrating astrocytomas of childhood
- (2013) K. J. Cohen et al. NEURO-ONCOLOGY
- Arsenic-induced interstitial myocardial fibrosis reveals a new insight into drug-induced long QT syndrome
- (2012) Wenfeng Chu et al. CARDIOVASCULAR RESEARCH
- MiR-296-3p regulates cell growth and multi-drug resistance of human glioblastoma by targeting ether-à-go-go (EAG1)
- (2012) Yifeng Bai et al. EUROPEAN JOURNAL OF CANCER
- MicroRNA-133b inhibits the growth of non-small-cell lung cancer by targeting the epidermal growth factor receptor
- (2012) Lingxiang Liu et al. FEBS Journal
- Upregulation of microRNA-1 and microRNA-133 contributes to arsenic-induced cardiac electrical remodeling
- (2012) Hongli Shan et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
- Phase I study of arsenic trioxide and temozolomide in combination with radiation therapy in patients with malignant gliomas
- (2012) Sean A. Grimm et al. JOURNAL OF NEURO-ONCOLOGY
- MiR-133b Targets Antiapoptotic Genes and Enhances Death Receptor-Induced Apoptosis
- (2012) Juan P. Patron et al. PLoS One
- Exosome-Mediated Transfer of miR-133b from Multipotent Mesenchymal Stromal Cells to Neural Cells Contributes to Neurite Outgrowth
- (2012) Hongqi Xin et al. STEM CELLS
- The role of Eag and HERG channels in cell proliferation and apoptotic cell death in SK-OV-3 ovarian cancer cell line
- (2011) Viren Asher et al. Cancer Cell International
- Myocardial gene expression of microRNA-133a and myosin heavy and light chains, in conjunction with clinical parameters, predict regression of left ventricular hypertrophy after valve replacement in patients with aortic stenosis
- (2011) A. V. Villar et al. HEART
- MicroRNA-133b is a key promoter of cervical carcinoma development through the activation of the ERK and AKT1 pathways
- (2011) W Qin et al. ONCOGENE
- Transcriptional and Post-Transcriptional Mechanisms for Oncogenic Overexpression of Ether À Go-Go K+ Channel
- (2011) Huixian Lin et al. PLoS One
- Eag and HERG potassium channels as novel therapeutic targets in cancer
- (2011) Viren Asher et al. World Journal of Surgical Oncology
- MicroRNA-34a: A Novel Tumor Suppressor in p53-mutant Glioma Cell Line U251
- (2010) Shihai Luan et al. ARCHIVES OF MEDICAL RESEARCH
- MicroRNA-34a Inhibits Glioblastoma Growth by Targeting Multiple Oncogenes
- (2009) Y. Li et al. CANCER RESEARCH
- miR-124 and miR-137 inhibit proliferation of glioblastoma multiforme cells and induce differentiation of brain tumor stem cells
- (2008) Joachim Silber et al. BMC Medicine
- MicroRNA Regulation of Cell Lineages in Mouse and Human Embryonic Stem Cells
- (2008) Kathryn N. Ivey et al. Cell Stem Cell
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started