Intestine-specific expression ofMOGAT2partially restores metabolic efficiency inMogat2-deficient mice

Proof of mechanism for the DGAT1 inhibitor AZD7687: results from a first-time-in-human single-dose study

(2012) H. Denison et al.   DIABETES OBESITY & METABOLISM

Intestinal DGAT1 deficiency reduces postprandial triglyceride and retinyl ester excursions by inhibiting chylomicron secretion and delaying gastric emptying

(2012) Gene P. Ables et al.   JOURNAL OF LIPID RESEARCH

Mammalian Triacylglycerol Metabolism: Synthesis, Lipolysis, and Signaling

(2011) Rosalind A. Coleman et al.   CHEMICAL REVIEWS

Targeting Acyl-CoA:Diacylglycerol Acyltransferase 1 (DGAT1) with Small Molecule Inhibitors for the Treatment of Metabolic Diseases

(2011) Jingsong Cao et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Deficiency of MGAT2 increases energy expenditure without high-fat feeding and protects genetically obese mice from excessive weight gain

(2011) David W. Nelson et al.   JOURNAL OF LIPID RESEARCH

The Biogenesis of Chylomicrons

(2010) Charles M. Mansbach et al.   Annual Review of Physiology

Identification of Body Fat Mass as a Major Determinant of Metabolic Rate in Mice

(2010) K. J. Kaiyala et al.   DIABETES

A Recurring Problem With the Analysis of Energy Expenditure in Genetic Models Expressing Lean and Obese Phenotypes

(2010) A. A. Butler et al.   DIABETES

Intestine-specific expression of acyl CoA:diacylglycerol acyltransferase 1 reverses resistance to diet-induced hepatic steatosis and obesity inDgat1−/−mice

(2010) Bonggi Lee et al.   JOURNAL OF LIPID RESEARCH

Deficiency of the intestinal enzyme acyl CoA:monoacylglycerol acyltransferase-2 protects mice from metabolic disorders induced by high-fat feeding

(2009) Chi-Liang Eric Yen et al.   NATURE MEDICINE

Thematic Review Series: Glycerolipids.DGAT enzymes and triacylglycerol biosynthesis

(2008) Chi-Liang Eric Yen et al.   JOURNAL OF LIPID RESEARCH