DGAT1 deficiency decreases PPAR expression and does not lead to lipotoxicity in cardiac and skeletal muscle

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Changes in muscle mass with mechanical load: possible cellular mechanismsThis paper is one of a selection of papers published in this Special Issue, entitled 14th International Biochemistry of Exercise Conference – Muscles as Molecular and Metabolic Machines, and has undergone the Journal’s usual peer review process.

(2009) Espen E. Spangenburg   Applied Physiology Nutrition and Metabolism

Identification of a Physiologically Relevant Endogenous Ligand for PPARα in Liver

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Specific role for acyl CoA:Diacylglycerol acyltransferase 1 (Dgat1) in hepatic steatosis due to exogenous fatty acids

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DGAT1 Expression Increases Heart Triglyceride Content but Ameliorates Lipotoxicity

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Pathogenesis and Pathophysiology of the Cardiometabolic Syndrome

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Discovery of a Potent, Selective, and Orally Efficacious Pyrimidinooxazinyl Bicyclooctaneacetic Acid Diacylglycerol Acyltransferase-1 Inhibitor

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Adipose triglyceride lipase in human skeletal muscle is upregulated by exercise training

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