Myeloperoxidase and serum amyloid A contribute to impaired in vivo reverse cholesterol transport during the acute phase response but not group IIA secretory phospholipase A2

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Scavenger Receptor BI-mediated Selective Uptake Is Required for the Remodeling of High Density Lipoprotein by Endothelial Lipase

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Hepatic SR-BI, not endothelial lipase, expression determines biliary cholesterol secretion in mice,

(2009) Harmen Wiersma et al.   JOURNAL OF LIPID RESEARCH

HDL Remodeling During the Acute Phase Response

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Atherosclerosis in rheumatoid arthritis and systemic lupus erythematosus

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SR-BI protects against endotoxemia in mice through its roles in glucocorticoid production and hepatic clearance

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Myeloperoxidase, modified lipoproteins, and atherogenesis: Fig. 1.

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The role of reverse cholesterol transport in animals and humans and relationship to atherosclerosis

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SR-BI-mediated HDL cholesteryl ester delivery in the adrenal gland

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Translating molecular discoveries into new therapies for atherosclerosis

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Methionine oxidation impairs reverse cholesterol transport by apolipoprotein A-I

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