Fanca deficiency reduces A/T transitions in somatic hypermutation and alters class switch recombination junctions in mouse B cells
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Title
Fanca deficiency reduces A/T transitions in somatic hypermutation and alters class switch recombination junctions in mouse B cells
Authors
Keywords
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Journal
JOURNAL OF EXPERIMENTAL MEDICINE
Volume 211, Issue 6, Pages 1011-1018
Publisher
Rockefeller University Press
Online
2014-05-06
DOI
10.1084/jem.20131637
References
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- Fanconi anaemia and the repair of Watson and Crick DNA crosslinks
- (2013) Molly C. Kottemann et al. NATURE
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- (2013) Emilie Renaud et al. PLoS One
- Regulation of Rev1 by the Fanconi anemia core complex
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- Functional and physical interaction between the mismatch repair and FA-BRCA pathways
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- Preventing Nonhomologous End Joining Suppresses DNA Repair Defects of Fanconi Anemia
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- The Fanconi Anemia Core Complex Is Dispensable during Somatic Hypermutation and Class Switch Recombination
- (2010) Peter H. L. Krijger et al. PLoS One
- Ku70 Corrupts DNA Repair in the Absence of the Fanconi Anemia Pathway
- (2010) P. Pace et al. SCIENCE
- Dual Roles for DNA Polymerase Theta in Alternative End-Joining Repair of Double-Strand Breaks in Drosophila
- (2010) Sze Ham Chan et al. PLoS Genetics
- The FANC pathway and BLM collaborate during mitosis to prevent micro-nucleation and chromosome abnormalities
- (2009) Valeria Naim et al. NATURE CELL BIOLOGY
- Role of Mre11 in chromosomal nonhomologous end joining in mammalian cells
- (2009) Emilie Rass et al. NATURE STRUCTURAL & MOLECULAR BIOLOGY
- Balancing AID and DNA repair during somatic hypermutation
- (2009) Man Liu et al. TRENDS IN IMMUNOLOGY
- SHMTool: A webserver for comparative analysis of somatic hypermutation datasets
- (2008) Thomas MacCarthy et al. DNA REPAIR
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