miR-150 regulates the development of NK and iNKT cells

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A novel Ncr1-Cre mouse reveals the essential role of STAT5 for NK-cell survival and development

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Loss of microRNAs in thymus perturbs invariant NKT cell development and function

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Distinct Requirements of MicroRNAs in NK Cell Activation, Survival, and Function

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The transcription factor c-Myb primes CD4+CD8+ immature thymocytes for selection into the iNKT lineage

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Raising the NKT cell family

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Maturation of mouse NK cells is a 4-stage developmental program

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Dicer-Dependent MicroRNA Pathway Controls Invariant NKT Cell Development

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The gene encoding early growth response 2, a target of the transcription factor NFAT, is required for the development and maturation of natural killer T cells

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Tie2cre-induced inactivation of the miRNA-processing enzyme Dicer disrupts invariant NKT cell development

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MicroRNAs miR-186 and miR-150 Down-regulate Expression of the Pro-apoptotic Purinergic P2X7Receptor by Activation of Instability Sites at the 3′-Untranslated Region of the Gene That Decrease Steady-state Levels of the Transcript

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Dicer-dependent microRNA pathway safeguards regulatory T cell function

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Selective miRNA disruption in T reg cells leads to uncontrolled autoimmunity

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c-Myb Is an Evolutionary Conserved miR-150 Target and miR-150/c-Myb Interaction Is Important for Embryonic Development

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