Critical role of Bcl11b in suppressor function of T regulatory cells and prevention of inflammatory bowel disease
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Title
Critical role of Bcl11b in suppressor function of T regulatory cells and prevention of inflammatory bowel disease
Authors
Keywords
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Journal
JOURNAL OF EXPERIMENTAL MEDICINE
Volume 208, Issue 10, Pages 2069-2081
Publisher
Rockefeller University Press
Online
2011-08-30
DOI
10.1084/jem.20102683
References
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Note: Only part of the references are listed.- Antigen-specific clonal expansion and cytolytic effector function of CD8+T lymphocytes depend on the transcription factor Bcl11b
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- Foxo proteins cooperatively control the differentiation of Foxp3+ regulatory T cells
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- Reprogramming of T Cells to Natural Killer-Like Cells upon Bcl11b Deletion
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- BCL11B enhances TCR/CD28-triggered NF-κB activation through up-regulation of Cot kinase gene expression in T-lymphocytes
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- Regulatory T Cell-Derived Interleukin-10 Limits Inflammation at Environmental Interfaces
- (2008) Yuri P. Rubtsov et al. IMMUNITY
- A distal cis-regulatory element, CNS-9, controls NFAT1 and IRF4-mediated IL-10 gene activation in T helper cells
- (2008) Choong-Gu Lee et al. MOLECULAR IMMUNOLOGY
- BCL11B is a general transcriptional repressor of the HIV-1 long terminal repeat in T lymphocytes through recruitment of the NuRD complex
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