Altered VEGF-stimulated Ca2+ signaling in part underlies pregnancy-adapted eNOS activity in UAEC

The loss of sustained Ca2+ signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

(2013) Jennifer Krupp et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

[Ca2+]i signaling vs. eNOS expression as determinants of NO output in uterine artery endothelium: relative roles in pregnancy adaptation and reversal by VEGF165

(2011) Fu-Xian Yi et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Pregnancy Enhances Sustained Ca2+ Bursts and Endothelial Nitric Oxide Synthase Activation in Ovine Uterine Artery Endothelial Cells Through Increased Connexin 43 Function1

(2009) Fu-Xian Yi et al.   BIOLOGY OF REPRODUCTION

Critical role of TRPC6 channels in VEGF-mediated angiogenesis

(2009) Ruiliang Ge et al.   CANCER LETTERS

Effects of Combined Phosphorylation at Ser-617 and Ser-1179 in Endothelial Nitric-oxide Synthase on EC50(Ca2+) Values for Calmodulin Binding and Enzyme Activation

(2009) Quang-Kim Tran et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Vasodilator factors in the systemic and local adaptations to pregnancy

(2009) Gloria Valdes et al.   Reproductive Biology and Endocrinology

Vascular endothelial growth factor acts through novel, pregnancy-enhanced receptor signalling pathways to stimulate endothelial nitric oxide synthase activity in uterine artery endothelial cells

(2008) Mary A. Grummer et al.   BIOCHEMICAL JOURNAL