Delving into disability in Crohn's disease: Dysregulation of molecular pathways may explain skeletal muscle loss in Crohn's disease

Objectively measured muscle fatigue in Crohn's disease: Correlation with self-reported fatigue and associated factors for clinical application

(2013) D.R. van Langenberg et al.   Journal of Crohns & Colitis

Mitochondrial dysfunction, persistent oxidative damage, and catalase inhibition in immune cells of naïve and treated Crohnʼs disease

(2009) Belén Beltrán et al.   INFLAMMATORY BOWEL DISEASES

NDRG2, a novel regulator of myoblast proliferation, is regulated by anabolic and catabolic factors

(2009) Victoria C. Foletta et al.   JOURNAL OF PHYSIOLOGY-LONDON

IL-6 and Serum Amyloid A Synergy Mediates Angiotensin II-Induced Muscle Wasting

(2009) L. Zhang et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Involvement of phosphoinositide 3-kinase and Akt in the induction of muscle protein degradation by proteolysis-inducing factor

(2008) Steven T. Russell et al.   BIOCHEMICAL JOURNAL

Sarcopenia is prevalent in patients with Crohnʼs disease in clinical remission

(2008) Stéphane M. Schneider et al.   INFLAMMATORY BOWEL DISEASES

Skeletal muscle growth in young rats is inhibited by chronic exposure to IL-6 but preserved by concurrent voluntary endurance exercise

(2008) P. W. Bodell et al.   JOURNAL OF APPLIED PHYSIOLOGY

Human Sarcopenia Reveals an Increase in SOCS-3 and Myostatin and a Reduced Efficiency of Akt Phosphorylation

(2008) Bertrand Léger et al.   REJUVENATION RESEARCH