C3 glomerulopathy–associated CFHR1 mutation alters FHR oligomerization and complement regulation
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Title
C3 glomerulopathy–associated CFHR1 mutation alters FHR oligomerization and complement regulation
Authors
Keywords
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Journal
JOURNAL OF CLINICAL INVESTIGATION
Volume 123, Issue 6, Pages 2434-2446
Publisher
American Society for Clinical Investigation
Online
2013-05-24
DOI
10.1172/jci68280
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- Eculizumab in a Patient with Dense-Deposit Disease
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- (2012) Seetha Radhakrishnan et al. NEW ENGLAND JOURNAL OF MEDICINE
- Complement factor H variants I890 and L1007 while commonly associated with atypical hemolytic uremic syndrome are polymorphisms with no functional significance
- (2011) Agustín Tortajada et al. KIDNEY INTERNATIONAL
- Genome-wide association study identifies susceptibility loci for IgA nephropathy
- (2011) Ali G Gharavi et al. NATURE GENETICS
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- An imbalance of human complement regulatory proteins CFHR1, CFHR3 and factor H influences risk for age-related macular degeneration (AMD)
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- Human C3 mutation reveals a mechanism of dense deposit disease pathogenesis and provides insights into complement activation and regulation
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- Identification of a mutation in complement factor H-related protein 5 in patients of Cypriot origin with glomerulonephritis
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- Factor H-related protein 1 (CFHR-1) inhibits complement C5 convertase activity and terminal complex formation
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